Jinxuan Zhao
No. 321 Zhongshan Road
Nanjing, China
Nanjing, China
Last Updated: 09/26/2026
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Found 16 publications
Assessment of Coronary Flow Reserve in a Mouse Model of Heart Failure with Preserved Ejection Fraction Using Doppler Echocardiography.
Journal: Journal of visualized experiments : JoVE
Published: April 13, 2026
Correction: Intravenously delivered mesenchymal stem cells prevent microvascular obstruction formation after myocardial ischemia/reperfusion injury.
Journal: Basic research in cardiology
Published: April 09, 2026
Excessive accumulation of epicardial adipose tissue promotes microvascular obstruction formation after myocardial ischemia/reperfusion through modulating macrophages polarization.
Journal: Cardiovascular diabetology
Published: March 21, 2024
Dapagliflozin attenuates LPS-induced myocardial injury by reducing ferroptosis.
Journal: Journal of bioenergetics and biomembranes
Published: March 08, 2024
MSC-Derived Exosomes Mitigate Myocardial Ischemia/Reperfusion Injury by Reducing Neutrophil Infiltration and the Formation of Neutrophil Extracellular Traps.
Journal: International journal of nanomedicine
Published: September 19, 2023
Bone marrow mesenchymal stem cell-derived exosomes attenuate cardiac hypertrophy and fibrosis in pressure overload induced remodeling.
Journal: In vitro cellular & developmental biology. Animal
Published: March 25, 2020
Extracellular vesicles derived from Krüppel-Like Factor 2-overexpressing endothelial cells attenuate myocardial ischemia-reperfusion injury by preventing Ly6Chigh monocyte recruitment.
Journal: Theranostics
Published: February 29, 2020
Intravenously delivered mesenchymal stem cells prevent microvascular obstruction formation after myocardial ischemia/reperfusion injury.
Journal: Basic research in cardiology
Published: January 29, 2020
miRNA-181a over-expression in mesenchymal stem cell-derived exosomes influenced inflammatory response after myocardial ischemia-reperfusion injury.
Journal: Life sciences
Published: May 22, 2019
Trimethylamine N-Oxide Exacerbates Cardiac Fibrosis via Activating the NLRP3 Inflammasome.
Journal: Frontiers in physiology
Published: February 15, 2019
Long non-coding RNA MEG3 knockdown attenuates endoplasmic reticulum stress-mediated apoptosis by targeting p53 following myocardial infarction.
Journal: Journal of cellular and molecular medicine
Published: October 28, 2018
Last Updated: 09/26/2026