Jinying Wei

Shijiazhuang, China

Last Updated: 09/26/2026

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Found 14 publications

Irisin ameliorates UUO-induced renal interstitial fibrosis through TGF-β1/periostin/MMP-2 signaling pathway.

Irisin ameliorates UUO-induced renal interstitial fibrosis through TGF-β1/periostin/MMP-2 signaling pathway.

Journal: PloS one

Published: July 24, 2023

Irisin Ameliorates Renal Tubulointerstitial Fibrosis by Regulating the Smad4/β-Catenin Pathway in Diabetic Mice.

Irisin Ameliorates Renal Tubulointerstitial Fibrosis by Regulating the Smad4/β-Catenin Pathway in Diabetic Mice.

Journal: Diabetes, metabolic syndrome and obesity : targets and therapy

Published: March 02, 2023

PP2 Ameliorates Renal Fibrosis by Regulating the NF-κB/COX-2 and PPARγ/UCP2 Pathway in Diabetic Mice.

PP2 Ameliorates Renal Fibrosis by Regulating the NF-κB/COX-2 and PPARγ/UCP2 Pathway in Diabetic Mice.

Journal: Oxidative medicine and cellular longevity

Published: June 03, 2021

TXNIP deficiency mitigates podocyte apoptosis via restraining the activation of mTOR or p38 MAPK signaling in diabetic nephropathy.

TXNIP deficiency mitigates podocyte apoptosis via restraining the activation of mTOR or p38 MAPK signaling in diabetic nephropathy.

Journal: Experimental cell research

Published: July 24, 2019

Thioredoxin-interacting protein deficiency alleviates phenotypic alterations of podocytes via inhibition of mTOR activation in diabetic nephropathy.

Thioredoxin-interacting protein deficiency alleviates phenotypic alterations of podocytes via inhibition of mTOR activation in diabetic nephropathy.

Journal: Journal of cellular physiology

Published: November 12, 2018

Knockdown of NLRP3 alleviates high glucose or TGFB1-induced EMT in human renal tubular cells

Knockdown of NLRP3 alleviates high glucose or TGFB1-induced EMT in human renal tubular cells

Journal: Journal of molecular endocrinology

Published: October 12, 2018

Anthocyanins inhibit high glucose-induced renal tubular cell apoptosis caused by oxidative stress in db/db mice.

Anthocyanins inhibit high glucose-induced renal tubular cell apoptosis caused by oxidative stress in db/db mice.

Journal: International journal of molecular medicine

Published: June 12, 2017

SOCS-1 is involved in TNF-α-induced mitochondrial dysfunction and apoptosis in renal tubular epithelial cells.

SOCS-1 is involved in TNF-α-induced mitochondrial dysfunction and apoptosis in renal tubular epithelial cells.

Journal: Tissue & cell

Published: March 15, 2017

Thioredoxin-interacting protein regulates lipid metabolism via Akt/mTOR pathway in diabetic kidney disease.

Thioredoxin-interacting protein regulates lipid metabolism via Akt/mTOR pathway in diabetic kidney disease.

Journal: The international journal of biochemistry & cell biology

Published: April 20, 2016

CD36 is involved in high glucose-induced epithelial to mesenchymal transition in renal tubular epithelial cells.

CD36 is involved in high glucose-induced epithelial to mesenchymal transition in renal tubular epithelial cells.

Journal: Biochemical and biophysical research communications

Published: October 17, 2015

Anthocyanins inhibit high-glucose-induced cholesterol accumulation and inflammation by activating LXRα pathway in HK-2 cells.

Anthocyanins inhibit high-glucose-induced cholesterol accumulation and inflammation by activating LXRα pathway in HK-2 cells.

Journal: Drug design, development and therapy

Published: September 18, 2015

Inhibition of c-Src/p38 MAPK pathway ameliorates renal tubular epithelial cells apoptosis in db/db mice.

Inhibition of c-Src/p38 MAPK pathway ameliorates renal tubular epithelial cells apoptosis in db/db mice.

Journal: Molecular and cellular endocrinology

Published: May 19, 2015

Showing 1-12 of 14

Last Updated: 09/26/2026