Jiong Cui
Chazhong Road 20
Fuzhou, 350005, China
Fuzhou, 350005, China
Last Updated: 09/26/2026
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Found 22 publications
FBP1 Is Associated with Attenuated Mitochondrial Injury in Renal Tubular Epithelial Cells of Diabetic Kidney Disease via Modulation of Lactate Metabolism.
Journal: International journal of molecular sciences
Published: March 28, 2026
C3a/C3aR axis is involved in diabetic kidney injury by regulating podocyte mitophagy in diabetic nephropathy.
Journal: International journal of molecular medicine
Published: June 20, 2025
Clinical and Pathological Characteristics and Prognosis of Diabetic Nephropathy Patients With Tertiary Lymphoid Structures.
Journal: Journal of diabetes research
Published: January 05, 2025
KLF15 deficiency contributes complement activation and podocyte injury in IgA nephropathy via regulating p300/ NF-κB axis.
Journal: Experimental cell research
Published: January 04, 2025
Risk factors and predictive model for renal outcomes in autoimmune membranous nephropathy with and without acute kidney injury: a retrospective cohort study.
Journal: PeerJ
Published: October 18, 2024
Association of time-averaged serum uric acid level with clinicopathological information and long-term outcomes in patients with IgA nephropathy.
Journal: PeerJ
Published: November 24, 2023
C3a/C3aR synergies with TGF-β to promote epithelial-mesenchymal transition of renal tubular epithelial cells via the activation of the NLRP3 inflammasome.
Journal: Journal of translational medicine
Published: September 23, 2023
Chloroquine inhibits NLRP3 inflammasomes activation and alleviates renal fibrosis in mouse model of hyperuricemic nephropathy with aggravation by a high-fat-diet.
Journal: International immunopharmacology
Published: February 03, 2023
Time-Averaged Hematuria as a Prognostic Indicator of Renal Outcome in Patients with IgA Nephropathy.
Time-Averaged Hematuria as a Prognostic Indicator of Renal Outcome in Patients with IgA Nephropathy.
Journal: Journal of clinical medicine
Published: October 24, 2022
Complement C3 mediates podocyte injury through TLR4/NFΚB-P65 signaling during ischemia-reperfusion acute kidney injury and post-injury fibrosis.
Journal: European journal of medical research
Published: August 23, 2022
Last Updated: 09/26/2026