Joan Espin-Domingo

Lund, M 22184, Sweden

Last Updated: 09/26/2026

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Found 8 publications

ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKα2.

ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKα2.

Journal: Molecular metabolism

Published: October 02, 2019

ApoAI-derived peptide increases glucose tolerance and prevents formation of atherosclerosis in mice.

ApoAI-derived peptide increases glucose tolerance and prevents formation of atherosclerosis in mice.

Journal: Diabetologia

Published: September 18, 2018

Site-specific glycations of apolipoprotein A-I lead to differentiated functional effects on lipid-binding and on glucose metabolism.

Site-specific glycations of apolipoprotein A-I lead to differentiated functional effects on lipid-binding and on glucose metabolism.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: December 13, 2017

Structural determinants in ApoA-I amyloidogenic variants explain improved cholesterol metabolism despite low HDL levels.

Structural determinants in ApoA-I amyloidogenic variants explain improved cholesterol metabolism despite low HDL levels.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: June 01, 2017

Synchrotron radiation circular dichroism spectroscopy reveals structural divergences in HDL-bound apoA-I variants.

Synchrotron radiation circular dichroism spectroscopy reveals structural divergences in HDL-bound apoA-I variants.

Journal: Scientific reports

Published: April 12, 2017

Molecular crowding impacts the structure of apolipoprotein A-I with potential implications on in vivo metabolism and function.

Molecular crowding impacts the structure of apolipoprotein A-I with potential implications on in vivo metabolism and function.

Journal: Biopolymers

Published: November 25, 2015

Dual Actions of Apolipoprotein A-I on Glucose-Stimulated Insulin Secretion and Insulin-Independent Peripheral Tissue Glucose Uptake Lead to Increased Heart and Skeletal Muscle Glucose Disposal.

Dual Actions of Apolipoprotein A-I on Glucose-Stimulated Insulin Secretion and Insulin-Independent Peripheral Tissue Glucose Uptake Lead to Increased Heart and Skeletal Muscle Glucose Disposal.

Journal: Diabetes

Published: October 29, 2015

ApoA-I Milano stimulates lipolysis in adipose cells independently of cAMP/PKA activation.

ApoA-I Milano stimulates lipolysis in adipose cells independently of cAMP/PKA activation.

Journal: Journal of lipid research

Published: September 11, 2014

Showing 1-8 of 8

Last Updated: 09/26/2026