John H. Weiss

John H. Weiss

Neurology
101 The City Drive South
Orange, CA 92868

Last Updated: 09/26/2026

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Found 28 publications

Mechanisms of delayed ischemia/reperfusion evoked ROS generation in the hippocampal CA1 zone of adult mouse brain slices.

Mechanisms of delayed ischemia/reperfusion evoked ROS generation in the hippocampal CA1 zone of adult mouse brain slices.

Journal: Research square
Published: May 02, 2025

Mechanisms of delayed ischemia/reperfusion evoked ROS generation in the hippocampal CA1 zone of adult mouse brain slices.

Mechanisms of delayed ischemia/reperfusion evoked ROS generation in the hippocampal CA1 zone of adult mouse brain slices.

Journal: Scientific reports
Published: December 13, 2024

Skeletal muscle effects of antisense oligonucleotides targeting glycogen synthase 1 in a mouse model of Pompe disease.

Skeletal muscle effects of antisense oligonucleotides targeting glycogen synthase 1 in a mouse model of Pompe disease.

Journal: Clinical and translational medicine
Published: March 24, 2024

Skeletal muscle effects of antisense oligonucleotides targeting glycogen synthase 1 in a mouse model of Pompe disease.

Skeletal muscle effects of antisense oligonucleotides targeting glycogen synthase 1 in a mouse model of Pompe disease.

Journal: bioRxiv : the preprint server for biology
Published: March 11, 2024

Editorial: Excitotoxicity Turns 50. The Death That Never Dies.

Editorial: Excitotoxicity Turns 50. The Death That Never Dies.

Journal: Frontiers in neuroscience
Published: December 08, 2021

VCP/p97 inhibitor CB-5083 modulates muscle pathology in a mouse model of VCP inclusion body myopathy.

VCP/p97 inhibitor CB-5083 modulates muscle pathology in a mouse model of VCP inclusion body myopathy.

Journal: Journal of translational medicine
Published: August 21, 2021

Blocking Mitochondrial Zn2+ Accumulation after Ischemia Reduces Mitochondrial Dysfunction and Neuronal Injury.

Blocking Mitochondrial Zn2+ Accumulation after Ischemia Reduces Mitochondrial Dysfunction and Neuronal Injury.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: April 22, 2021

The Multifaceted Roles of Zinc in Neuronal Mitochondrial Dysfunction.

The Multifaceted Roles of Zinc in Neuronal Mitochondrial Dysfunction.

Journal: Biomedicines
Published: February 25, 2021

Zn2+ entry through the mitochondrial calcium uniporter is a critical contributor to mitochondrial dysfunction and neurodegeneration.

Zn2+ entry through the mitochondrial calcium uniporter is a critical contributor to mitochondrial dysfunction and neurodegeneration.

Journal: Experimental neurology
Published: August 15, 2019

Rapid Intramitochondrial Zn2+ Accumulation in CA1 Hippocampal Pyramidal Neurons After Transient Global Ischemia: A Possible Contributor to Mitochondrial Disruption and Cell Death.

Rapid Intramitochondrial Zn2+ Accumulation in CA1 Hippocampal Pyramidal Neurons After Transient Global Ischemia: A Possible Contributor to Mitochondrial Disruption and Cell Death.

Journal: Journal of neuropathology and experimental neurology
Published: June 01, 2019

Mitochondrial Zn2+ Accumulation: A Potential Trigger of Hippocampal Ischemic Injury.

Mitochondrial Zn2+ Accumulation: A Potential Trigger of Hippocampal Ischemic Injury.

Journal: The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry
Published: May 11, 2018

Zn2+-induced disruption of neuronal mitochondrial function: Synergism with Ca2+, critical dependence upon cytosolic Zn2+ buffering, and contributions to neuronal injury.

Zn2+-induced disruption of neuronal mitochondrial function: Synergism with Ca2+, critical dependence upon cytosolic Zn2+ buffering, and contributions to neuronal injury.

Journal: Experimental neurology
Published: October 28, 2017
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Last Updated: 09/26/2026