Jonathan M. Alexander

Cardiology

21 South St
Ridgefield, CT 06877

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 13 publications

Reframing Efficiency: How Public Deliberation Strengthens Public Health Policy.

Reframing Efficiency: How Public Deliberation Strengthens Public Health Policy.

Journal: American journal of public health

Published: July 23, 2026

Use of polyadenosine tail mimetics to enhance mRNA expression from genes associated with haploinsufficiency disorders.

Use of polyadenosine tail mimetics to enhance mRNA expression from genes associated with haploinsufficiency disorders.

Journal: Molecular therapy. Nucleic acids

Published: August 08, 2024

Inhibition of GSK3α,β rescues cognitive phenotypes in a preclinical mouse model of CTNNB1 syndrome.

Inhibition of GSK3α,β rescues cognitive phenotypes in a preclinical mouse model of CTNNB1 syndrome.

Journal: EMBO molecular medicine

Published: July 18, 2023

Excessive β-Catenin in Excitatory Neurons Results in Reduced Social and Increased Repetitive Behaviors and Altered Expression of Multiple Genes Linked to Human Autism.

Excessive β-Catenin in Excitatory Neurons Results in Reduced Social and Increased Repetitive Behaviors and Altered Expression of Multiple Genes Linked to Human Autism.

Journal: Frontiers in synaptic neuroscience

Published: September 27, 2019

Learning impairments and molecular changes in the brain caused by β-catenin loss.

Learning impairments and molecular changes in the brain caused by β-catenin loss.

Journal: Human molecular genetics

Published: March 07, 2019

Rac1 Activity Is Modulated by Huntingtin and Dysregulated in Models of Huntington's Disease.

Rac1 Activity Is Modulated by Huntingtin and Dysregulated in Models of Huntington's Disease.

Journal: Journal of Huntington's disease

Published: December 31, 2018

Huntingtin associates with the actin cytoskeleton and α-actinin isoforms to influence stimulus dependent morphology changes.

Huntingtin associates with the actin cytoskeleton and α-actinin isoforms to influence stimulus dependent morphology changes.

Journal: PloS one

Published: August 15, 2018

Social Stimulus Causes Aberrant Activation of the Medial Prefrontal Cortex in a Mouse Model With Autism-Like Behaviors.

Social Stimulus Causes Aberrant Activation of the Medial Prefrontal Cortex in a Mouse Model With Autism-Like Behaviors.

Journal: Frontiers in synaptic neuroscience

Published: May 31, 2018

Retracted: Elevated NADPH oxidase activity contributes to oxidative stress and cell death in Huntington's disease.

Retracted: Elevated NADPH oxidase activity contributes to oxidative stress and cell death in Huntington's disease.

Journal: Human molecular genetics

Published: October 04, 2017

APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures.

APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures.

Journal: Neurobiology of disease

Published: August 19, 2016

Elevated NADPH oxidase activity contributes to oxidative stress and cell death in Huntington's disease.

Elevated NADPH oxidase activity contributes to oxidative stress and cell death in Huntington's disease.

Journal: Human molecular genetics

Published: December 11, 2012

Reagents that block neuronal death from Huntington's disease also curb oxidative stress.

Reagents that block neuronal death from Huntington's disease also curb oxidative stress.

Journal: Neuroreport

Published: November 03, 2011

Showing 1-12 of 13

Last Updated: 09/26/2026