Jose Alvarez-Rodriguez
Madrid, MD 28029, Spain
Last Updated: 09/26/2026
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Found 19 publications
Corrigendum to "Beta-hydroxybutyrate counteracts the deleterious effects of a saturated high-fat diet on synaptic AMPAR receptors and cognitive performance" [Mol Metabol (2025) 102207].
Journal: Molecular metabolism
Published: June 25, 2026
Presenilin-dependent regulation of neuronal tau pathology via the autophagy and proteasome pathways.
Presenilin-dependent regulation of neuronal tau pathology via the autophagy and proteasome pathways.
Journal: Acta neuropathologica communications
Published: October 29, 2025
Beta-hydroxybutyrate counteracts the deleterious effects of a saturated high-fat diet on synaptic AMPAR receptors and cognitive performance.
Journal: Molecular metabolism
Published: April 14, 2025
Nr4a2 blocks oAβ-mediated synaptic plasticity dysfunction and ameliorates spatial memory deficits in the APP Sw,Ind mouse.
Journal: bioRxiv : the preprint server for biology
Published: February 08, 2024
Activity-Dependent Nr4a2 Induction Modulates Synaptic Expression of AMPA Receptors and Plasticity via a Ca2+/CRTC1/CREB Pathway.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: July 08, 2022
Nr4a2 Transcription Factor in Hippocampal Synaptic Plasticity, Memory and Cognitive Dysfunction: A Perspective Review.
Journal: Frontiers in molecular neuroscience
Published: September 29, 2021
microRNAs as Early Biomarkers of Alzheimer's Disease: A Synaptic Perspective.
Journal: Cells
Published: November 29, 2020
Sensing of nutrients by CPT1C controls SAC1 activity to regulate AMPA receptor trafficking.
Journal: The Journal of cell biology
Published: December 09, 2019
Proteasomal-Mediated Degradation of AKAP150 Accompanies AMPAR Endocytosis during cLTD.
Journal: eNeuro
Published: June 06, 2019
Altered microRNAs related to synaptic function as potential plasma biomarkers for Alzheimer's disease.
Journal: Alzheimer's research & therapy
Published: February 13, 2019
Blood-brain barrier dysfunction underlying Alzheimer's disease is induced by an SSAO/VAP-1-dependent cerebrovascular activation with enhanced Aβ deposition.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: November 20, 2018
Last Updated: 09/26/2026