Josu Rapun
Madrid, MD, Spain
Last Updated: 09/26/2026
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Found 8 publications
Propionic Acidemia-Induced Proarrhythmic Electrophysiological Alterations in Human iPSC-Derived Cardiomyocytes.
Journal: Journal of inherited metabolic disease
Published: January 08, 2025
Two concurrent mechanisms are responsible for the INa increase produced by dapagliflozin and empagliflozin in healthy and heart failure cardiomyocytes.
Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: December 19, 2024
Reply to Benndorff and DiFrancesco: Reliable human HCN4 single-channel recordings using the cell-attached configuration in expression systems.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 08, 2024
Variable Penetrance and Expressivity of a Rare Pore Loss-of-Function Mutation (p.L889V) of Nav1.5 Channels in Three Spanish Families.
Journal: International journal of molecular sciences
Published: February 27, 2024
A gain-of-function HCN4 mutant in the HCN domain is responsible for inappropriate sinus tachycardia in a Spanish family.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: November 30, 2023
Empagliflozin and Dapagliflozin Increase Na+ and Inward Rectifier K+ Current Densities in Human Cardiomyocytes Derived from Induced Pluripotent Stem Cells (hiPSC-CMs).
Journal: Cells
Published: September 29, 2022
Regulation of cardiac ion channels by transcription factors: Looking for new opportunities of druggable targets for the treatment of arrhythmias.
Journal: Biochemical pharmacology
Published: June 27, 2022
A Cantú syndrome mutation produces dual effects on KATP channels by disrupting ankyrin B regulation.
A Cantú syndrome mutation produces dual effects on KATP channels by disrupting ankyrin B regulation.
Journal: The Journal of general physiology
Published: July 08, 2021
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Last Updated: 09/26/2026