Juanjuan Shi

State Key Laboratory Of Systems Medicine For Cancer
Shanghai, China

Last Updated: 09/26/2026

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Found 19 publications

LY6D identifies persistent stem-like cells driving pancreatic tumourigenesis.

LY6D identifies persistent stem-like cells driving pancreatic tumourigenesis.

Journal: Gut

Published: July 23, 2025

The diverse pancreatic tumor cell-intrinsic response to IFNγ is determined by epigenetic heterogeneity.

The diverse pancreatic tumor cell-intrinsic response to IFNγ is determined by epigenetic heterogeneity.

Journal: Cancer letters

Published: January 02, 2023

Tumor Cell-Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis.

Tumor Cell-Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: May 18, 2022

Glycolysis addiction compensating for a defective pentose phosphate pathway confers gemcitabine sensitivity in SETD2-deficient pancreatic cancer.

Glycolysis addiction compensating for a defective pentose phosphate pathway confers gemcitabine sensitivity in SETD2-deficient pancreatic cancer.

Journal: Biochemical and biophysical research communications

Published: April 30, 2022

Interleukin-37 protects against acinar cell pyroptosis in acute pancreatitis.

Interleukin-37 protects against acinar cell pyroptosis in acute pancreatitis.

Journal: JCI insight

Published: April 22, 2022

Ketogenesis acts as an endogenous protective programme to restrain inflammatory macrophage activation during acute pancreatitis.

Ketogenesis acts as an endogenous protective programme to restrain inflammatory macrophage activation during acute pancreatitis.

Journal: EBioMedicine

Published: November 18, 2021

SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer.

SULF2 enhances GDF15-SMAD axis to facilitate the initiation and progression of pancreatic cancer.

Journal: Cancer letters

Published: November 15, 2021

Acinar cell NLRP3 inflammasome and gasdermin D (GSDMD) activation mediates pyroptosis and systemic inflammation in acute pancreatitis.

Acinar cell NLRP3 inflammasome and gasdermin D (GSDMD) activation mediates pyroptosis and systemic inflammation in acute pancreatitis.

Journal: British journal of pharmacology

Published: July 18, 2020

Macrophage phenotypic switch orchestrates the inflammation and repair/regeneration following acute pancreatitis injury.

Macrophage phenotypic switch orchestrates the inflammation and repair/regeneration following acute pancreatitis injury.

Journal: EBioMedicine

Published: January 23, 2020

Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarcinoma.

Perineural Invasion Reprograms the Immune Microenvironment through Cholinergic Signaling in Pancreatic Ductal Adenocarcinoma.

Journal: Cancer research

Published: August 28, 2019

Inflammation and development of pancreatic ductal adenocarcinoma.

Inflammation and development of pancreatic ductal adenocarcinoma.

Journal: Chinese clinical oncology

Published: March 13, 2019

Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis.

Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis.

Journal: Gut

Published: January 24, 2019

Showing 1-12 of 19

Last Updated: 09/26/2026