Julia Bolik

Kiel, SH 24118, Germany

Last Updated: 09/26/2026

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Found 8 publications

Tetraspanin 8 Subfamily Members Regulate Substrate-Specificity of a Disintegrin and Metalloprotease 17.

Tetraspanin 8 Subfamily Members Regulate Substrate-Specificity of a Disintegrin and Metalloprotease 17.

Journal: Cells
Published: July 20, 2022

Inhibition of ADAM17 impairs endothelial cell necroptosis and blocks metastasis.

Inhibition of ADAM17 impairs endothelial cell necroptosis and blocks metastasis.

Journal: The Journal of experimental medicine
Published: May 21, 2020

Cell-autonomous hepatocyte-specific GP130 signaling is sufficient to trigger a robust innate immune response in mice.

Cell-autonomous hepatocyte-specific GP130 signaling is sufficient to trigger a robust innate immune response in mice.

Journal: Journal of hepatology
Published: February 22, 2020

Gut microbiota shape 'inflamm-ageing' cytokines and account for age-dependent decline in DNA damage repair.

Gut microbiota shape 'inflamm-ageing' cytokines and account for age-dependent decline in DNA damage repair.

Journal: Gut
Published: February 17, 2019

Early heme oxygenase 1 induction delays tumour initiation and enhances DNA damage repair in liver macrophages of Mdr2-/- mice.

Early heme oxygenase 1 induction delays tumour initiation and enhances DNA damage repair in liver macrophages of Mdr2-/- mice.

Journal: Scientific reports
Published: May 17, 2018

The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.

The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.

Journal: Oncotarget
Published: November 11, 2017

ADAM17 is required for EGF-R-induced intestinal tumors via IL-6 trans-signaling.

ADAM17 is required for EGF-R-induced intestinal tumors via IL-6 trans-signaling.

Journal: The Journal of experimental medicine
Published: September 13, 2017

IL-6 trans-signaling is essential for the development of hepatocellular carcinoma in mice.

IL-6 trans-signaling is essential for the development of hepatocellular carcinoma in mice.

Journal: Hepatology (Baltimore, Md.)
Published: May 25, 2016
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Last Updated: 09/26/2026