Julie Dunys
Valbonne, France
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 9 publications
Dipeptidyl peptidase 4 contributes to Alzheimer's disease-like defects in a mouse model and is increased in sporadic Alzheimer's disease brains.
Journal: The Journal of biological chemistry
Published: December 13, 2020
Aminopeptidase A contributes to biochemical, anatomical and cognitive defects in Alzheimer's disease (AD) mouse model and is increased at early stage in sporadic AD brain.
Journal: Acta neuropathologica
Published: December 01, 2020
Are N- and C-terminally truncated Aβ species key pathological triggers in Alzheimer's disease?
Journal: The Journal of biological chemistry
Published: August 26, 2018
Nuclear p53-mediated repression of autophagy involves PINK1 transcriptional down-regulation.
Journal: Cell death and differentiation
Published: February 13, 2017
β-Amyloid Precursor Protein Intracellular Domain Controls Mitochondrial Function by Modulating Phosphatase and Tensin Homolog-Induced Kinase 1 Transcription in Cells and in Alzheimer Mice Models.
Journal: Biological psychiatry
Published: August 26, 2016
Aβ42 oligomers modulate β-secretase through an XBP-1s-dependent pathway involving HRD1.
Journal: Scientific reports
Published: August 10, 2016
The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer model.
Journal: Molecular psychiatry
Published: March 24, 2016
The transcription factor X-box binding protein-1 in neurodegenerative diseases.
Journal: Molecular neurodegeneration
Published: July 31, 2014
The β-secretase-derived C-terminal fragment of βAPP, C99, but not Aβ, is a key contributor to early intraneuronal lesions in triple-transgenic mouse hippocampus.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: November 16, 2012
Showing 1-9 of 9
Last Updated: 09/26/2026