Jun-chao Wu

Jiangsu Key Laboratory Of Neuropsychiatric Diseases
Suzhou, 215123, China

Last Updated: 09/26/2026

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Found 24 publications

Cobrotoxin suppresses TLR4-NF-κB/STAT1 signaling to mitigate glia-mediated neuroinflammation and motor deficits in Parkinson's disease.

Cobrotoxin suppresses TLR4-NF-κB/STAT1 signaling to mitigate glia-mediated neuroinflammation and motor deficits in Parkinson's disease.

Journal: International immunopharmacology

Published: July 10, 2026

Fat on Fire: Disrupted Microglial Lipid Metabolism as a Driver of Anesthetic Neurotoxicity.

Fat on Fire: Disrupted Microglial Lipid Metabolism as a Driver of Anesthetic Neurotoxicity.

Journal: Neuroscience bulletin

Published: October 07, 2025

TIGAR overexpression alleviates intracerebral hemorrhage injury in mice by suppressing ATF4/NOX4/p22phox-mediated oxidative stress and inflammation.

TIGAR overexpression alleviates intracerebral hemorrhage injury in mice by suppressing ATF4/NOX4/p22phox-mediated oxidative stress and inflammation.

Journal: Acta pharmacologica Sinica

Published: August 20, 2025

NADPH acts as an endogenous P2X7 receptor modulator to gate neuroinflammatory responses of microglia.

NADPH acts as an endogenous P2X7 receptor modulator to gate neuroinflammatory responses of microglia.

Journal: Acta pharmacologica Sinica

Published: March 14, 2025

The P2X7 receptor mediates NADPH transport across the plasma membrane.

The P2X7 receptor mediates NADPH transport across the plasma membrane.

Journal: Biochemical and biophysical research communications

Published: June 24, 2024

Regulation of FSP1 myristoylation by NADPH: A novel mechanism for ferroptosis inhibition.

Regulation of FSP1 myristoylation by NADPH: A novel mechanism for ferroptosis inhibition.

Journal: Redox biology

Published: April 16, 2024

Nicotinamide riboside restores nicotinamide adenine dinucleotide levels and alleviates brain injury by inhibiting oxidative stress and neuroinflammation in a mouse model of intracerebral hemorrhage.

Nicotinamide riboside restores nicotinamide adenine dinucleotide levels and alleviates brain injury by inhibiting oxidative stress and neuroinflammation in a mouse model of intracerebral hemorrhage.

Journal: Molecular neurobiology

Published: October 04, 2023

RRx-001 Exerts Neuroprotection Against LPS-Induced Microglia Activation and Neuroinflammation Through Disturbing the TLR4 Pathway.

RRx-001 Exerts Neuroprotection Against LPS-Induced Microglia Activation and Neuroinflammation Through Disturbing the TLR4 Pathway.

Journal: Frontiers in pharmacology

Published: March 04, 2022

TIGAR plays neuroprotective roles in KA-induced excitotoxicity through reducing neuroinflammation and improving mitochondrial function.

TIGAR plays neuroprotective roles in KA-induced excitotoxicity through reducing neuroinflammation and improving mitochondrial function.

Journal: Neurochemistry international

Published: May 04, 2021

Long-term running exercise alleviates cognitive dysfunction in APP/PSEN1 transgenic mice via enhancing brain lysosomal function.

Long-term running exercise alleviates cognitive dysfunction in APP/PSEN1 transgenic mice via enhancing brain lysosomal function.

Journal: Acta pharmacologica Sinica

Published: January 20, 2021

NADPH is superior to NADH or edaravone in ameliorating metabolic disturbance and brain injury in ischemic stroke.

NADPH is superior to NADH or edaravone in ameliorating metabolic disturbance and brain injury in ischemic stroke.

Journal: Acta pharmacologica Sinica

Published: January 07, 2021

NADPH and Mito-Apocynin Treatment Protects Against KA-Induced Excitotoxic Injury Through Autophagy Pathway.

NADPH and Mito-Apocynin Treatment Protects Against KA-Induced Excitotoxic Injury Through Autophagy Pathway.

Journal: Frontiers in cell and developmental biology

Published: September 30, 2020

Showing 1-12 of 24

Last Updated: 09/26/2026