Jun-ichi Hayashi

Tsukuba, 3058572, Japan

Last Updated: 09/26/2026

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Found 32 publications

Aberrant RNA processing contributes to the pathogenesis of mitochondrial diseases in trans-mitochondrial mouse model carrying mitochondrial tRNALeu(UUR) with a pathogenic A2748G mutation.

Aberrant RNA processing contributes to the pathogenesis of mitochondrial diseases in trans-mitochondrial mouse model carrying mitochondrial tRNALeu(UUR) with a pathogenic A2748G mutation.

Journal: Nucleic acids research
Published: January 17, 2022

Mitochondrial DNA deletion-dependent podocyte injuries in Mito-miceΔ, a murine model of mitochondrial disease.

Mitochondrial DNA deletion-dependent podocyte injuries in Mito-miceΔ, a murine model of mitochondrial disease.

Journal: Experimental animals
Published: July 29, 2021

A high mutation load of m.14597A>G in MT-ND6 causes Leigh syndrome.

A high mutation load of m.14597A>G in MT-ND6 causes Leigh syndrome.

Journal: Scientific reports
Published: February 20, 2021

Disruption of the mouse Shmt2 gene confers embryonic anaemia via foetal liver-specific metabolomic disorders.

Disruption of the mouse Shmt2 gene confers embryonic anaemia via foetal liver-specific metabolomic disorders.

Journal: Scientific reports
Published: July 09, 2019

Mito-mice∆ and mitochondrial DNA mutator mice as models of human osteoporosis caused not by aging but by hyperparathyroidism.

Mito-mice∆ and mitochondrial DNA mutator mice as models of human osteoporosis caused not by aging but by hyperparathyroidism.

Journal: Experimental animals
Published: July 06, 2018

Propofol induces a metabolic switch to glycolysis and cell death in a mitochondrial electron transport chain-dependent manner.

Propofol induces a metabolic switch to glycolysis and cell death in a mitochondrial electron transport chain-dependent manner.

Journal: PloS one
Published: November 20, 2017

Cytoplasmic transfer of heritable elements other than mtDNA from SAMP1 mice into mouse tumor cells suppresses their ability to form tumors in C57BL6 mice.

Cytoplasmic transfer of heritable elements other than mtDNA from SAMP1 mice into mouse tumor cells suppresses their ability to form tumors in C57BL6 mice.

Journal: Biochemical and biophysical research communications
Published: September 06, 2017

Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethal.

Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethal.

Journal: Scientific reports
Published: August 15, 2017

Association of predicted pathogenic mutations in mitochondrial ND genes with distant metastasis in NSCLC and colon cancer.

Association of predicted pathogenic mutations in mitochondrial ND genes with distant metastasis in NSCLC and colon cancer.

Journal: Scientific reports
Published: July 18, 2017

A novel mutation in TAZ causes mitochondrial respiratory chain disorder without cardiomyopathy.

A novel mutation in TAZ causes mitochondrial respiratory chain disorder without cardiomyopathy.

Journal: Journal of human genetics
Published: May 20, 2016

Mutations in mitochondrial DNA regulate mitochondrial diseases and metastasis but do not regulate aging.

Mutations in mitochondrial DNA regulate mitochondrial diseases and metastasis but do not regulate aging.

Journal: Current opinion in genetics & development
Published: December 14, 2015

Corrigendum: Epigenetic regulation of the nuclear-coded GCAT and SHMT2 genes confers human age-associated mitochondrial respiration defects.

Corrigendum: Epigenetic regulation of the nuclear-coded GCAT and SHMT2 genes confers human age-associated mitochondrial respiration defects.

Journal: Scientific reports
Published: October 06, 2015
Showing 1-12 of 32

Last Updated: 09/26/2026