Justin You

Toronto, ON, Canada

Last Updated: 09/26/2026

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Found 8 publications

Protocol for autofluorescence removal in microglia by photobleaching in free-floating immunofluorescent staining of mouse brain tissue.

Protocol for autofluorescence removal in microglia by photobleaching in free-floating immunofluorescent staining of mouse brain tissue.

Journal: STAR protocols

Published: August 03, 2025

Interventionally targeting somatic CAG expansions can be a rapid disease-modifying therapeutic avenue: Preclinical evidence.

Interventionally targeting somatic CAG expansions can be a rapid disease-modifying therapeutic avenue: Preclinical evidence.

Journal: bioRxiv : the preprint server for biology

Published: May 07, 2025

Knockout of Dectin-1 does not modify disease onset or progression in a MATR3 S85C knock-in mouse model of ALS.

Knockout of Dectin-1 does not modify disease onset or progression in a MATR3 S85C knock-in mouse model of ALS.

Journal: Heliyon

Published: May 10, 2024

MATR3 pathogenic variants differentially impair its cryptic splicing repression function.

MATR3 pathogenic variants differentially impair its cryptic splicing repression function.

Journal: FEBS letters

Published: December 06, 2023

Microglia and Astrocytes in Amyotrophic Lateral Sclerosis: Disease-Associated States, Pathological Roles, and Therapeutic Potential.

Microglia and Astrocytes in Amyotrophic Lateral Sclerosis: Disease-Associated States, Pathological Roles, and Therapeutic Potential.

Journal: Biology

Published: August 11, 2023

Evidence of Metabolic Dysfunction in Amyotrophic Lateral Sclerosis (ALS) Patients and Animal Models.

Evidence of Metabolic Dysfunction in Amyotrophic Lateral Sclerosis (ALS) Patients and Animal Models.

Journal: Biomolecules

Published: April 21, 2023

Selective Loss of MATR3 in Spinal Interneurons, Upper Motor Neurons and Hippocampal CA1 Neurons in a MATR3 S85C Knock-In Mouse Model of Amyotrophic Lateral Sclerosis.

Selective Loss of MATR3 in Spinal Interneurons, Upper Motor Neurons and Hippocampal CA1 Neurons in a MATR3 S85C Knock-In Mouse Model of Amyotrophic Lateral Sclerosis.

Journal: Biology

Published: December 21, 2021

MATR3 F115C knock-in mice do not exhibit motor defects or neuropathological features of ALS.

MATR3 F115C knock-in mice do not exhibit motor defects or neuropathological features of ALS.

Journal: Biochemical and biophysical research communications

Published: May 10, 2021

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Last Updated: 09/26/2026