Kanhaiya L. Kumawat
Manesar, HR 122052, India
Last Updated: 09/26/2026
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Found 11 publications
Correction: Bispidine-Amino Acid Conjugates Act as a Novel Scaffold for the Design of Antivirals That Block Japanese Encephalitis Virus Replication.
Journal: PLoS neglected tropical diseases
Published: January 25, 2024
Japanese Encephalitis Virus infection increases USP42 to stabilize TRIM21 and OAS1 for neuroinflammatory and anti-viral response in human microglia.
Journal: Virology
Published: February 16, 2022
The host microRNA miR-301a blocks the IRF1-mediated neuronal innate immune response to Japanese encephalitis virus infection.
Journal: Science signaling
Published: February 16, 2017
Japanese Encephalitis Virus exploits the microRNA-432 to regulate the expression of Suppressor of Cytokine Signaling (SOCS) 5.
Journal: Scientific reports
Published: January 30, 2016
miR-146a suppresses cellular immune response during Japanese encephalitis virus JaOArS982 strain infection in human microglial cells.
Journal: Journal of neuroinflammation
Published: August 18, 2014
Regulatory role of TRIM21 in the type-I interferon pathway in Japanese encephalitis virus-infected human microglial cells.
Journal: Journal of neuroinflammation
Published: November 21, 2013
Interleukin-1β orchestrates underlying inflammatory responses in microglia via Krüppel-like factor 4.
Journal: Journal of neurochemistry
Published: June 25, 2013
Bispidine-amino acid conjugates act as a novel scaffold for the design of antivirals that block Japanese encephalitis virus replication.
Journal: PLoS neglected tropical diseases
Published: May 17, 2012
Therapeutic targeting of Krüppel-like factor 4 abrogates microglial activation.
Journal: Journal of neuroinflammation
Published: December 15, 2011
NLRP3 inflammasome: key mediator of neuroinflammation in murine Japanese encephalitis.
Journal: PloS one
Published: November 01, 2011
Showing 1-11 of 11
Last Updated: 09/26/2026