Karen G. Montgomery

St. Andrew's Place
Melbourne, VIC 3002, Australia

Last Updated: 09/26/2026

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Found 9 publications

PI3K activation in neural stem cells drives tumorigenesis which can be ameliorated by targeting the cAMP response element binding protein.

PI3K activation in neural stem cells drives tumorigenesis which can be ameliorated by targeting the cAMP response element binding protein.

Journal: Neuro-oncology

Published: May 03, 2018

Identification of Pik3ca Mutation as a Genetic Driver of Prostate Cancer That Cooperates with Pten Loss to Accelerate Progression and Castration-Resistant Growth.

Identification of Pik3ca Mutation as a Genetic Driver of Prostate Cancer That Cooperates with Pten Loss to Accelerate Progression and Castration-Resistant Growth.

Journal: Cancer discovery

Published: August 15, 2017

Inhibiting the system xC-/glutathione axis selectively targets cancers with mutant-p53 accumulation.

Inhibiting the system xC-/glutathione axis selectively targets cancers with mutant-p53 accumulation.

Journal: Nature communications

Published: November 15, 2016

Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis.

Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis.

Journal: Oncotarget

Published: September 06, 2016

Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling.

Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling.

Journal: Nature communications

Published: April 20, 2016

APR-246 potently inhibits tumour growth and overcomes chemoresistance in preclinical models of oesophageal adenocarcinoma.

APR-246 potently inhibits tumour growth and overcomes chemoresistance in preclinical models of oesophageal adenocarcinoma.

Journal: Gut

Published: April 14, 2015

Heterozygous expression of the oncogenic Pik3ca(H1047R) mutation during murine development results in fatal embryonic and extraembryonic defects.

Heterozygous expression of the oncogenic Pik3ca(H1047R) mutation during murine development results in fatal embryonic and extraembryonic defects.

Journal: Developmental biology

Published: January 25, 2015

Physiological expression of the PI3K-activating mutation Pik3ca(H1047R) combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice.

Physiological expression of the PI3K-activating mutation Pik3ca(H1047R) combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice.

Journal: The Biochemical journal

Published: December 11, 2013

Physiological levels of Pik3ca(H1047R) mutation in the mouse mammary gland results in ductal hyperplasia and formation of ERα-positive tumors.

Physiological levels of Pik3ca(H1047R) mutation in the mouse mammary gland results in ductal hyperplasia and formation of ERα-positive tumors.

Journal: PloS one

Published: March 09, 2012

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Last Updated: 09/26/2026