Katarina Willen
Lund, M, Sweden
Last Updated: 09/26/2026
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Found 12 publications
Multiple system atrophy-derived α-synuclein fibrils are more potent and display oligodendrocyte cell tropism compared to Parkinson's disease-derived fibrils.
Journal: Neurobiology of disease
Published: May 10, 2026
α-Synuclein overexpression increases the tropism of pre-formed fibrils and MSA-patient derived seeds towards oligodendroglia.
Journal: Acta neuropathologica communications
Published: February 04, 2026
Multiple system atrophy-associated oligodendroglial protein p25α stimulates formation of novel α-synuclein strain with enhanced neurodegenerative potential.
Journal: Acta neuropathologica
Published: October 26, 2020
APP depletion alters selective pre- and post-synaptic proteins.
Journal: Molecular and cellular neurosciences
Published: November 09, 2018
Heterogeneous Association of Alzheimer's Disease-Linked Amyloid-β and Amyloid-β Protein Precursor with Synapses.
Journal: Journal of Alzheimer's disease : JAD
Published: September 05, 2017
Aβ accumulation causes MVB enlargement and is modelled by dominant negative VPS4A.
Journal: Molecular neurodegeneration
Published: February 06, 2017
Pre-plaque conformational changes in Alzheimer's disease-linked Aβ and APP.
Journal: Nature communications
Published: May 31, 2016
Direct High Affinity Interaction between Aβ42 and GSK3α Stimulates Hyperphosphorylation of Tau. A New Molecular Link in Alzheimer's Disease?
Journal: ACS chemical neuroscience
Published: December 01, 2015
ESCRTs regulate amyloid precursor protein sorting in multivesicular bodies and intracellular amyloid-β accumulation.
Journal: Journal of cell science
Published: February 18, 2015
The inside-out amyloid hypothesis and synapse pathology in Alzheimer's disease.
Journal: Neuro-degenerative diseases
Published: May 16, 2013
Showing 1-12 of 12
Last Updated: 09/26/2026