Ke Cai
MOE Shanghai Key Laboratory Of Children's
Shanghai, 200092, China
Shanghai, 200092, China
Last Updated: 09/26/2026
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Found 23 publications
Adolescent high fructose consumption induces cardiac dysfunction in adulthood via elevated histone acetylation.
Journal: Translational pediatrics
Published: October 20, 2025
G3BP1 Succinylation at K413 is Critical for Cardiac Function by Modulating PI3K-AKT-mTOR Signal Axis.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: October 08, 2025
High fructose levels inhibit the proliferation of cardiomyocytes via the Notch1 signaling pathway.
Journal: Cellular signalling
Published: October 11, 2024
Branched-Chain Amino Acids Deficiency Promotes Diabetic Neuropathic Pain Through Upregulating LAT1 and Inhibiting Kv1.2 Channel.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: February 27, 2024
Upregulation of miR-21-5p rescues the inhibition of cardiomyocyte proliferation induced by high glucose through negative regulation of Rhob.
Journal: Journal of developmental origins of health and disease
Published: December 11, 2023
Maternal folic acid over-supplementation impairs cardiac function in mice offspring by inhibiting SOD1 expression.
Journal: Cardiovascular research
Published: December 05, 2023
Proteome profiling of early gestational plasma reveals novel biomarkers of congenital heart disease.
Proteome profiling of early gestational plasma reveals novel biomarkers of congenital heart disease.
Journal: EMBO molecular medicine
Published: April 06, 2023
NOTCH1 mitochondria localization during heart development promotes mitochondrial metabolism and the endothelial-to-mesenchymal transition in mice.
Journal: Nature communications
Published: July 01, 2022
Last Updated: 09/26/2026