Kenneth Y. Ting

Toronto, ON, Canada

Last Updated: 09/26/2026

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Found 15 publications

Intracellular accumulation of free cholesterol in macrophages triggers a PARP1 response to DNA damage and PARP1 impairs lipopolysaccharide-induced inflammatory response.

Intracellular accumulation of free cholesterol in macrophages triggers a PARP1 response to DNA damage and PARP1 impairs lipopolysaccharide-induced inflammatory response.

Journal: PloS one

Published: July 09, 2024

Measuring the rate of NADPH consumption by glutathione reductase in the cytosol and mitochondria.

Measuring the rate of NADPH consumption by glutathione reductase in the cytosol and mitochondria.

Journal: PloS one

Published: May 10, 2024

John Yudkin's hypothesis: sugar is a major dietary culprit in the development of cardiovascular disease.

John Yudkin's hypothesis: sugar is a major dietary culprit in the development of cardiovascular disease.

Journal: Frontiers in nutrition

Published: March 26, 2024

Revisiting the role of hypoxia-inducible factors and nuclear factor erythroid 2-related factor 2 in regulating macrophage inflammation and metabolism.

Revisiting the role of hypoxia-inducible factors and nuclear factor erythroid 2-related factor 2 in regulating macrophage inflammation and metabolism.

Journal: Frontiers in cellular and infection microbiology

Published: March 20, 2024

Cholesterol accumulation impairs HIF-1α-dependent immunometabolic reprogramming of LPS-stimulated macrophages by upregulating the NRF2 pathway.

Cholesterol accumulation impairs HIF-1α-dependent immunometabolic reprogramming of LPS-stimulated macrophages by upregulating the NRF2 pathway.

Journal: Scientific reports

Published: February 03, 2024

Fructose overconsumption-induced reprogramming of microglia metabolism and function.

Fructose overconsumption-induced reprogramming of microglia metabolism and function.

Journal: Frontiers in immunology

Published: January 23, 2024

Fructose-induced metabolic reprogramming of cancer cells.

Fructose-induced metabolic reprogramming of cancer cells.

Journal: Frontiers in immunology

Published: January 23, 2024

The neurorepellent SLIT2 inhibits LPS-induced proinflammatory signaling in macrophages.

The neurorepellent SLIT2 inhibits LPS-induced proinflammatory signaling in macrophages.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: November 20, 2023

Oxidized Low-Density Lipoprotein Accumulation in Macrophages Impairs Lipopolysaccharide-Induced Activation of AKT2, ATP Citrate Lyase, Acetyl-Coenzyme A Production, and Inflammatory Gene H3K27 Acetylation.

Oxidized Low-Density Lipoprotein Accumulation in Macrophages Impairs Lipopolysaccharide-Induced Activation of AKT2, ATP Citrate Lyase, Acetyl-Coenzyme A Production, and Inflammatory Gene H3K27 Acetylation.

Journal: ImmunoHorizons

Published: November 06, 2023

The multi-faceted role of NADPH in regulating inflammation in activated myeloid cells.

The multi-faceted role of NADPH in regulating inflammation in activated myeloid cells.

Journal: Frontiers in immunology

Published: October 26, 2023

Oxidized Low-Density Lipoprotein Accumulation Suppresses Glycolysis and Attenuates the Macrophage Inflammatory Response by Diverting Transcription from the HIF-1α to the Nrf2 Pathway.

Oxidized Low-Density Lipoprotein Accumulation Suppresses Glycolysis and Attenuates the Macrophage Inflammatory Response by Diverting Transcription from the HIF-1α to the Nrf2 Pathway.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: May 01, 2023

The PKN1- TRAF1 signaling axis as a potential new target for chronic lymphocytic leukemia.

The PKN1- TRAF1 signaling axis as a potential new target for chronic lymphocytic leukemia.

Journal: Oncoimmunology

Published: September 30, 2021

Showing 1-12 of 15

Last Updated: 09/26/2026