Kewarin Jinawong

Chiang Mai, 50200, Thailand

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 11 publications

Pharmacological inhibition of apoptosis, necroptosis, and ferroptosis confers effective cardioprotection in post-myocardial infarction in rats.

Pharmacological inhibition of apoptosis, necroptosis, and ferroptosis confers effective cardioprotection in post-myocardial infarction in rats.

Journal: Scientific reports

Published: June 11, 2025

Chronic mitochondrial dynamic-targeted therapy alleviates left ventricular dysfunction by reducing multiple programmed cell death in post-myocardial infarction rats.

Chronic mitochondrial dynamic-targeted therapy alleviates left ventricular dysfunction by reducing multiple programmed cell death in post-myocardial infarction rats.

Journal: European journal of pharmacology

Published: April 09, 2024

Modulating Mitochondrial Dynamics Mitigates Cognitive Impairment in Rats with Myocardial Infarction.

Modulating Mitochondrial Dynamics Mitigates Cognitive Impairment in Rats with Myocardial Infarction.

Journal: Current neuropharmacology

Published: June 13, 2023

Treatment with apoptosis inhibitor restores cognitive impairment in rats with myocardial infarction.

Treatment with apoptosis inhibitor restores cognitive impairment in rats with myocardial infarction.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: June 01, 2023

GSDMD-mediated pyroptosis dominantly promotes left ventricular remodeling and dysfunction in post-myocardial infarction: a comparison across modes of programmed cell death and mitochondrial involvement.

GSDMD-mediated pyroptosis dominantly promotes left ventricular remodeling and dysfunction in post-myocardial infarction: a comparison across modes of programmed cell death and mitochondrial involvement.

Journal: Journal of translational medicine

Published: October 19, 2022

Mild Cognitive impairment Occurs in Rats During the Early Remodeling Phase of Myocardial Infarction.

Mild Cognitive impairment Occurs in Rats During the Early Remodeling Phase of Myocardial Infarction.

Journal: Neuroscience

Published: February 10, 2022

Necrostatin-1 reduces cardiac and mitochondrial dysfunction in prediabetic rats.

Necrostatin-1 reduces cardiac and mitochondrial dysfunction in prediabetic rats.

Journal: The Journal of endocrinology

Published: June 07, 2021

Cognitive impairment in myocardial infarction and heart failure.

Cognitive impairment in myocardial infarction and heart failure.

Journal: Acta physiologica (Oxford, England)

Published: November 27, 2020

Not only metformin, but also D-allulose, alleviates metabolic disturbance and cognitive decline in prediabetic rats.

Not only metformin, but also D-allulose, alleviates metabolic disturbance and cognitive decline in prediabetic rats.

Journal: Nutritional neuroscience

Published: November 05, 2020

D-allulose provides cardioprotective effect by attenuating cardiac mitochondrial dysfunction in obesity-induced insulin-resistant rats.

D-allulose provides cardioprotective effect by attenuating cardiac mitochondrial dysfunction in obesity-induced insulin-resistant rats.

Journal: European journal of nutrition

Published: June 27, 2020

Necrostatin-1 Mitigates Cognitive Dysfunction in Prediabetic Rats With No Alteration in Insulin Sensitivity.

Necrostatin-1 Mitigates Cognitive Dysfunction in Prediabetic Rats With No Alteration in Insulin Sensitivity.

Journal: Diabetes

Published: November 11, 2019

Showing 1-11 of 11

Last Updated: 09/26/2026