Kira Meyerovich

Brussels, BRU, Belgium

Last Updated: 09/26/2026

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Found 9 publications

SKAP2, a Candidate Gene for Type 1 Diabetes, Regulates β-Cell Apoptosis and Glycemic Control in Newly Diagnosed Patients.

SKAP2, a Candidate Gene for Type 1 Diabetes, Regulates β-Cell Apoptosis and Glycemic Control in Newly Diagnosed Patients.

Journal: Diabetes
Published: January 29, 2020

The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes.

The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes.

Journal: Journal of molecular endocrinology
Published: April 17, 2018

Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis.

Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis.

Journal: Cell death & disease
Published: July 24, 2017

MCL-1 Is a Key Antiapoptotic Protein in Human and Rodent Pancreatic β-Cells.

MCL-1 Is a Key Antiapoptotic Protein in Human and Rodent Pancreatic β-Cells.

Journal: Diabetes
Published: October 14, 2016

Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation.

Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation.

Journal: Journal of molecular endocrinology
Published: March 25, 2016

A20 Inhibits β-Cell Apoptosis by Multiple Mechanisms and Predicts Residual β-Cell Function in Type 1 Diabetes.

A20 Inhibits β-Cell Apoptosis by Multiple Mechanisms and Predicts Residual β-Cell Function in Type 1 Diabetes.

Journal: Molecular endocrinology (Baltimore, Md.)
Published: December 15, 2015

A20 deficiency sensitizes pancreatic beta cells to cytokine-induced apoptosis in vitro but does not influence type 1 diabetes development in vivo.

A20 deficiency sensitizes pancreatic beta cells to cytokine-induced apoptosis in vitro but does not influence type 1 diabetes development in vivo.

Journal: Cell death & disease
Published: October 16, 2015

The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro.

The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro.

Journal: Diabetologia
Published: July 24, 2015

Heterozygous inactivation of plasma membrane Ca(2+)-ATPase in mice increases glucose-induced insulin release and beta cell proliferation, mass and viability.

Heterozygous inactivation of plasma membrane Ca(2+)-ATPase in mice increases glucose-induced insulin release and beta cell proliferation, mass and viability.

Journal: Diabetologia
Published: March 26, 2015
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Last Updated: 09/26/2026