Kosei Ishimaru
Fukuoka, Japan
Last Updated: 09/26/2026
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Found 8 publications
Excessive HIF-1α driven by phospholipid metabolism causes septic cardiomyopathy through cytopathic hypoxia.
Journal: Nature cardiovascular research
Published: August 12, 2024
Deferasirox Targeting Ferroptosis Synergistically Ameliorates Myocardial Ischemia Reperfusion Injury in Conjunction With Cyclosporine A.
Journal: Journal of the American Heart Association
Published: December 29, 2023
Cardiac Autoantibodies Against Cardiac Troponin I in Post-Myocardial Infarction Heart Failure: Evaluation in a Novel Murine Model and Applications in Therapeutics.
Journal: Circulation. Heart failure
Published: July 31, 2023
Doxorubicin causes ferroptosis and cardiotoxicity by intercalating into mitochondrial DNA and disrupting Alas1-dependent heme synthesis.
Journal: Science signaling
Published: November 01, 2022
Iron Overload via Heme Degradation in the Endoplasmic Reticulum Triggers Ferroptosis in Myocardial Ischemia-Reperfusion Injury.
Journal: JACC. Basic to translational science
Published: February 03, 2022
Ethoxyquin is a Competent Radical-Trapping Antioxidant for Preventing Ferroptosis in Doxorubicin Cardiotoxicity.
Journal: Journal of cardiovascular pharmacology
Published: January 07, 2022
Excessive Hypoxia-Inducible Factor-1α Expression Induces Cardiac Rupture via p53-Dependent Apoptosis After Myocardial Infarction.
Journal: Journal of the American Heart Association
Published: September 02, 2021
Heart Rate Reduction with Ivabradine Prevents Cardiac Rupture after Myocardial Infarction in Mice.
Journal: Cardiovascular drugs and therapy
Published: December 07, 2020
Showing 1-8 of 8
Last Updated: 09/26/2026