Kostas Paschos

London, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 11 publications

Evaluation of the contribution of the minor envelope complex glycoprotein 3 ectodomain to the porcine reproductive and respiratory syndrome virus 1 neutralizing antibody response.

Evaluation of the contribution of the minor envelope complex glycoprotein 3 ectodomain to the porcine reproductive and respiratory syndrome virus 1 neutralizing antibody response.

Journal: The Journal of general virology
Published: September 03, 2026

Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C.

Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C.

Journal: Nucleic acids research
Published: September 14, 2018

The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.

The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.

Journal: Pathogens (Basel, Switzerland)
Published: January 30, 2018

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

Journal: Journal of virology
Published: November 03, 2017

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

Journal: PLoS biology
Published: January 12, 2017

Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.

Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.

Journal: Nucleic acids research
Published: August 02, 2016

Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.

Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.

Journal: The Journal of experimental medicine
Published: January 25, 2016

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

Journal: PLoS pathogens
Published: October 12, 2015

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Journal: PLoS pathogens
Published: January 30, 2015

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Journal: PLoS pathogens
Published: November 02, 2012

BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV.

BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV.

Journal: Nucleic acids research
Published: May 16, 2012
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Last Updated: 09/26/2026