Kuo-cheng Lan
TPQ, Taiwan
Last Updated: 09/26/2026
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Found 31 publications
Functional interaction between advanced glycation end-products and IGFBP-3 contributes to renal pathology in aging-associated chronic kidney disease.
Journal: Tissue & cell
Published: January 11, 2026
Advanced glycation end product-its receptor axis participates in myoblast senescence in vitro and delayed muscle regeneration of senescence/aging skeletal muscles in vivo.
Journal: Archives of biochemistry and biophysics
Published: October 21, 2025
Gut Microbiota-Linked Benefits of Low-Intensity Pulsed Ultrasound Rejuvenate the Ageing Muscle.
Journal: Journal of cachexia, sarcopenia and muscle
Published: September 18, 2025
Polycyclic aromatic hydrocarbons-enriched diesel exhaust particles induced hypertrophy in rat primary neonatal cardiomyocytes and apoptotic cell death in H9c2 cardiomyocytes.
Journal: Toxicology
Published: August 25, 2025
Glucagon-like peptide-1 receptor (GLP-1R) agonists prevent tributyltin-induced muscle atrophy/wasting via restoring GLP-1R signaling in vitro and in mice.
Journal: Ecotoxicology and environmental safety
Published: May 20, 2025
Uremic toxin indoxyl sulfate induces the imbalance of cellular iron metabolism and oxidative stress to interfere with myogenesis in myoblasts.
Journal: Chemico-biological interactions
Published: February 23, 2025
Nε-(1-Carboxymethyl)-L-lysine/RAGE Signaling Drives Metastasis and Cancer Stemness through ERK/NFκB axis in Osteosarcoma.
Journal: International journal of biological sciences
Published: October 06, 2023
Inhibition of Indoxyl Sulfate-Induced Reactive Oxygen Species-Related Ferroptosis Alleviates Renal Cell Injury In Vitro and Chronic Kidney Disease Progression In Vivo.
Journal: Antioxidants (Basel, Switzerland)
Published: August 30, 2023
Nε-(1-Carboxymethyl)-L-lysine, an advanced glycation end product, exerts malignancy on chondrosarcoma via the activation of cancer stemness.
Journal: Archives of toxicology
Published: January 04, 2023
A ubiquitous endocrine disruptor tributyltin induces muscle wasting and retards muscle regeneration.
A ubiquitous endocrine disruptor tributyltin induces muscle wasting and retards muscle regeneration.
Journal: Journal of cachexia, sarcopenia and muscle
Published: May 10, 2022
Last Updated: 09/26/2026