Kurt A. Krobert
Oslo, Norway
Last Updated: 09/26/2026
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Found 17 publications
RGS3L allows for an M2 muscarinic receptor-mediated RhoA-dependent inotropy in cardiomyocytes.
Journal: Basic research in cardiology
Published: January 18, 2021
Knockout of adenylyl cyclase isoform 5 or 6 differentially modifies the β1-adrenoceptor-mediated inotropic response.
Journal: Journal of molecular and cellular cardiology
Published: March 07, 2019
Constitutive inhibitory G protein activity upon adenylyl cyclase-dependent cardiac contractility is limited to adenylyl cyclase type 6.
Journal: PloS one
Published: January 31, 2019
Hypothermia elongates the contraction-relaxation cycle in explanted human failing heart decreasing the time for ventricular filling during diastole.
Journal: American journal of physiology. Heart and circulatory physiology
Published: July 14, 2018
Identification of essential residues for binding and activation in the human 5-HT7(a) serotonin receptor by molecular modeling and site-directed mutagenesis.
Journal: Frontiers in behavioral neuroscience
Published: January 30, 2015
The inotropic effect of the active metabolite of levosimendan, OR-1896, is mediated through inhibition of PDE3 in rat ventricular myocardium.
Journal: PloS one
Published: June 30, 2014
Non-classical regulation of β1- and β 2-adrenoceptor-mediated inotropic responses in rat heart ventricle by the G protein Gi.
Journal: Naunyn-Schmiedeberg's archives of pharmacology
Published: June 03, 2014
The atypical antipsychotics clozapine and olanzapine promote down-regulation and display functional selectivity at human 5-HT7 receptors.
Journal: British journal of pharmacology
Published: May 21, 2014
Gi proteins regulate adenylyl cyclase activity independent of receptor activation.
Journal: PloS one
Published: April 15, 2014
NSC23766, a widely used inhibitor of Rac1 activation, additionally acts as a competitive antagonist at muscarinic acetylcholine receptors.
Journal: The Journal of pharmacology and experimental therapeutics
Published: July 27, 2013
Connective tissue growth factor/CCN2 attenuates β-adrenergic receptor responsiveness and cardiotoxicity by induction of G protein-coupled receptor kinase-5 in cardiomyocytes.
Journal: Molecular pharmacology
Published: June 20, 2013
Last Updated: 09/26/2026