Kurt Gleich

Heidelberg, VIC 3084, Australia

Last Updated: 09/26/2026

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Found 13 publications

The AMPK activator ATX-304 alters cellular metabolism to protect against cisplatin-induced acute kidney injury.

The AMPK activator ATX-304 alters cellular metabolism to protect against cisplatin-induced acute kidney injury.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: January 05, 2024

Blocking AMPK signalling to acetyl-CoA carboxylase increases cisplatin-induced acute kidney injury and suppresses the benefit of metformin.

Blocking AMPK signalling to acetyl-CoA carboxylase increases cisplatin-induced acute kidney injury and suppresses the benefit of metformin.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: April 20, 2022

Mutation of regulatory phosphorylation sites in PFKFB2 does not affect the anti-fibrotic effect of metformin in the kidney.

Mutation of regulatory phosphorylation sites in PFKFB2 does not affect the anti-fibrotic effect of metformin in the kidney.

Journal: PloS one

Published: November 25, 2021

Mutation of regulatory phosphorylation sites in PFKFB2 worsens renal fibrosis.

Mutation of regulatory phosphorylation sites in PFKFB2 worsens renal fibrosis.

Journal: Scientific reports

Published: April 02, 2020

Phosphorylation of Acetyl-CoA Carboxylase by AMPK Reduces Renal Fibrosis and Is Essential for the Anti-Fibrotic Effect of Metformin.

Phosphorylation of Acetyl-CoA Carboxylase by AMPK Reduces Renal Fibrosis and Is Essential for the Anti-Fibrotic Effect of Metformin.

Journal: Journal of the American Society of Nephrology : JASN

Published: January 16, 2018

The Thiazide-Sensitive Co-Transporter Promotes the Development of Sodium Retention in Mice with Diet-Induced Obesity.

The Thiazide-Sensitive Co-Transporter Promotes the Development of Sodium Retention in Mice with Diet-Induced Obesity.

Journal: Kidney & blood pressure research

Published: September 03, 2015

Novel mechanisms of Na+ retention in obesity: phosphorylation of NKCC2 and regulation of SPAK/OSR1 by AMPK.

Novel mechanisms of Na+ retention in obesity: phosphorylation of NKCC2 and regulation of SPAK/OSR1 by AMPK.

Journal: American journal of physiology. Renal physiology

Published: May 09, 2014

Activation of AMPK reduces the co-transporter activity of NKCC1.

Activation of AMPK reduces the co-transporter activity of NKCC1.

Journal: Molecular membrane biology

Published: April 08, 2014

Absence of the lysosomal protein Limp-2 attenuates renal injury in crescentic glomerulonephritis.

Absence of the lysosomal protein Limp-2 attenuates renal injury in crescentic glomerulonephritis.

Journal: Immunology and cell biology

Published: November 05, 2013

AMPK couples plasma renin to cellular metabolism by phosphorylation of ACC1.

AMPK couples plasma renin to cellular metabolism by phosphorylation of ACC1.

Journal: American journal of physiology. Renal physiology

Published: June 21, 2013

Abnormal Processing of Autophagosomes in Transformed B Lymphocytes from SCARB2-Deficient Subjects.

Abnormal Processing of Autophagosomes in Transformed B Lymphocytes from SCARB2-Deficient Subjects.

Journal: BioResearch open access

Published: March 22, 2013

Limited capacity of proximal tubular proteolysis in mice with proteinuria.

Limited capacity of proximal tubular proteolysis in mice with proteinuria.

Journal: American journal of physiology. Renal physiology

Published: January 25, 2013

Showing 1-12 of 13

Last Updated: 09/26/2026