Laura Gennaccaro

Bologna, 40126, Italy

Last Updated: 09/26/2026

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Found 19 publications

Striatin knock out induces a gain of function of INa and impaired Ca2+ handling in mESC-derived cardiomyocytes.

Striatin knock out induces a gain of function of INa and impaired Ca2+ handling in mESC-derived cardiomyocytes.

Journal: Acta physiologica (Oxford, England)

Published: June 13, 2023

Cardiac Functional and Structural Abnormalities in a Mouse Model of CDKL5 Deficiency Disorder.

Cardiac Functional and Structural Abnormalities in a Mouse Model of CDKL5 Deficiency Disorder.

Journal: International journal of molecular sciences

Published: February 08, 2023

Expression of a Secretable, Cell-Penetrating CDKL5 Protein Enhances the Efficacy of Gene Therapy for CDKL5 Deficiency Disorder.

Expression of a Secretable, Cell-Penetrating CDKL5 Protein Enhances the Efficacy of Gene Therapy for CDKL5 Deficiency Disorder.

Journal: Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics

Published: August 27, 2022

Luteolin Treatment Ameliorates Brain Development and Behavioral Performance in a Mouse Model of CDKL5 Deficiency Disorder.

Luteolin Treatment Ameliorates Brain Development and Behavioral Performance in a Mouse Model of CDKL5 Deficiency Disorder.

Journal: International journal of molecular sciences

Published: July 04, 2022

Treatment with a GSK-3β/HDAC Dual Inhibitor Restores Neuronal Survival and Maturation in an In Vitro and In Vivo Model of CDKL5 Deficiency Disorder.

Treatment with a GSK-3β/HDAC Dual Inhibitor Restores Neuronal Survival and Maturation in an In Vitro and In Vivo Model of CDKL5 Deficiency Disorder.

Journal: International journal of molecular sciences

Published: April 22, 2021

Inhibition of microglia overactivation restores neuronal survival in a mouse model of CDKL5 deficiency disorder.

Inhibition of microglia overactivation restores neuronal survival in a mouse model of CDKL5 deficiency disorder.

Journal: Journal of neuroinflammation

Published: February 03, 2021

A GABAB receptor antagonist rescues functional and structural impairments in the perirhinal cortex of a mouse model of CDKL5 deficiency disorder.

A GABAB receptor antagonist rescues functional and structural impairments in the perirhinal cortex of a mouse model of CDKL5 deficiency disorder.

Journal: Neurobiology of disease

Published: November 17, 2020

Age-Related Cognitive and Motor Decline in a Mouse Model of CDKL5 Deficiency Disorder is Associated with Increased Neuronal Senescence and Death.

Age-Related Cognitive and Motor Decline in a Mouse Model of CDKL5 Deficiency Disorder is Associated with Increased Neuronal Senescence and Death.

Journal: Aging and disease

Published: May 05, 2020

Correction to: Increased DNA Damage and Apoptosis in CDKL5-Deficient Neurons.

Correction to: Increased DNA Damage and Apoptosis in CDKL5-Deficient Neurons.

Journal: Molecular neurobiology

Published: February 18, 2020

Increased DNA Damage and Apoptosis in CDKL5-Deficient Neurons.

Increased DNA Damage and Apoptosis in CDKL5-Deficient Neurons.

Journal: Molecular neurobiology

Published: September 20, 2019

Pharmacotherapy with sertraline rescues brain development and behavior in a mouse model of CDKL5 deficiency disorder.

Pharmacotherapy with sertraline rescues brain development and behavior in a mouse model of CDKL5 deficiency disorder.

Journal: Neuropharmacology

Published: May 10, 2019

CDKL5 deficiency predisposes neurons to cell death through the deregulation of SMAD3 signaling.

CDKL5 deficiency predisposes neurons to cell death through the deregulation of SMAD3 signaling.

Journal: Brain pathology (Zurich, Switzerland)

Published: December 05, 2018

Showing 1-12 of 19

Last Updated: 09/26/2026