Laurent Palacios-Gagnoux
Nice, France
Last Updated: 09/26/2026
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Found 8 publications
Fas acts as a T cell co-stimulatory receptor via both death domain-independent and -dependent mechanisms.
Journal: Cell death and differentiation
Published: July 04, 2025
PRIMA-1MET/APR-246 Can Partially Rescue In Vitro Cell Adhesion of Patient-Derived Junctional Epidermolysis Bullosa Epidermal Cells.
Journal: The Journal of investigative dermatology
Published: March 23, 2023
Optineurin links Hace1-dependent Rac ubiquitylation to integrin-mediated mechanotransduction to control bacterial invasion and cell division.
Journal: Nature communications
Published: September 24, 2021
Cell death for maintaining epithelial homeostasis
Journal: Medecine sciences : M/S
Published: December 18, 2019
Cell polarity and adherens junction formation inhibit epithelial Fas cell death receptor signaling.
Journal: The Journal of cell biology
Published: May 14, 2018
The Btk-dependent PIP5K1γ lipid kinase activation by Fas counteracts FasL-induced cell death.
Journal: Apoptosis : an international journal on programmed cell death
Published: September 08, 2017
Fas palmitoylation by the palmitoyl acyltransferase DHHC7 regulates Fas stability.
Journal: Cell death and differentiation
Published: April 09, 2014
Confluence switch signaling regulates ECM composition and the plasmin proteolytic cascade in keratinocytes.
Journal: Journal of cell science
Published: May 30, 2012
Showing 1-8 of 8
Last Updated: 09/26/2026