Li An
Key Laboratory Of Environmental Stress And Chronic Disease Control And Prevention Ministry Of Education
Shenyang, 110122, China
Shenyang, 110122, China
Last Updated: 09/26/2026
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Found 19 publications
The Mechanism of GABA in Attenuating Neuroinflammation in Alzheimer's Disease: CP/CEBPα/miR-34a-Mediated Suppression of HDAC2/3 in Astrocytes.
Journal: Foods (Basel, Switzerland)
Published: January 20, 2026
Histone Methylation, Energy Metabolism, and Alzheimer's Disease.
Journal: Aging and disease
Published: July 26, 2024
The molecular mechanism of γ-aminobutyric acid against AD: the role of CEBPα/circAPLP2/miR-671-5p in regulating CNTN1/2 expression.
Journal: Food & function
Published: February 03, 2023
Exploration of the Molecular Mechanism for Lipoprotein Lipase Expression Variations in SH-SY5Y Cells Exposed to Different Doses of Amyloid-Beta Protein.
Journal: Frontiers in aging neuroscience
Published: February 07, 2020
β-Hydroxybutyrate Ameliorates Aβ-Induced Downregulation of TrkA Expression by Inhibiting HDAC1/3 in SH-SY5Y Cells.
Journal: American journal of Alzheimer's disease and other dementias
Published: October 26, 2019
As a Histone Deacetylase Inhibitor, γ-Aminobutyric Acid Upregulates GluR2 Expression: An In Vitro and In Vivo Study.
Journal: Molecular nutrition & food research
Published: January 01, 2019
Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate.
Journal: Frontiers in cellular neuroscience
Published: August 26, 2017
Intermittent Fasting Protects against Alzheimer's Disease Possible through Restoring Aquaporin-4 Polarity.
Journal: Frontiers in molecular neuroscience
Published: July 07, 2017
Pretreatment of MQA, a caffeoylquinic acid derivative compound, protects against H2O2-induced oxidative stress in SH-SY5Y cells.
Journal: Neurological research
Published: November 02, 2016
Compound MQA, a Caffeoylquinic Acid Derivative, Protects Against NMDA-Induced Neurotoxicity and Potential Mechanisms In Vitro.
Journal: CNS neuroscience & therapeutics
Published: February 10, 2015
Sulforaphane ameliorates neurobehavioral deficits and protects the brain from amyloid β deposits and peroxidation in mice with Alzheimer-like lesions.
Journal: American journal of Alzheimer's disease and other dementias
Published: July 16, 2014
Last Updated: 09/26/2026