Li Lin

Nanning, China

Last Updated: 09/26/2026

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Found 12 publications

Reference intervals for erythrocyte parameters and hemoglobin electrophoresis parameters for young children in Guangxi.

Reference intervals for erythrocyte parameters and hemoglobin electrophoresis parameters for young children in Guangxi.

Journal: International journal of laboratory hematology

Published: March 18, 2022

Molecular Characterization of α- and β-Thalassaemia Among Children From 1 to 10 Years of Age in Guangxi, A Multi-Ethnic Region in Southern China.

Molecular Characterization of α- and β-Thalassaemia Among Children From 1 to 10 Years of Age in Guangxi, A Multi-Ethnic Region in Southern China.

Journal: Frontiers in pediatrics

Published: June 12, 2021

A tetranucleotide deletion in the ANK1 gene causes hereditary spherocytosis; a case of misdiagnosis.

A tetranucleotide deletion in the ANK1 gene causes hereditary spherocytosis; a case of misdiagnosis.

Journal: Gene

Published: September 02, 2019

Identification of a Novel 9.7 kb Deletion Causing α0-Thalassemia in Two Pregnant Women in Southern China.

Identification of a Novel 9.7 kb Deletion Causing α0-Thalassemia in Two Pregnant Women in Southern China.

Journal: Hemoglobin

Published: March 14, 2019

First Identification of the 3.5 kb Deletion (NC_000011.10: g.5224302-5227791del3490bp) on the β-Globin Gene Cluster in a Chinese Family.

First Identification of the 3.5 kb Deletion (NC_000011.10: g.5224302-5227791del3490bp) on the β-Globin Gene Cluster in a Chinese Family.

Journal: Hemoglobin

Published: January 08, 2019

Characterization of Hb Bart's Hydrops Fetalis Caused by - -SEA and a Large Novel α0-Thalassemia Deletion.

Characterization of Hb Bart's Hydrops Fetalis Caused by - -SEA and a Large Novel α0-Thalassemia Deletion.

Journal: Hemoglobin

Published: March 02, 2018

Characterization of a Large Novel α-Globin Gene Cluster Deletion Causing α0-Thalassemia in a Chinese Family.

Characterization of a Large Novel α-Globin Gene Cluster Deletion Causing α0-Thalassemia in a Chinese Family.

Journal: Hemoglobin

Published: November 23, 2017

The prevalence and molecular characterization of (δβ)0 -thalassemia and hereditary persistence of fetal hemoglobin in the Chinese Zhuang population.

The prevalence and molecular characterization of (δβ)0 -thalassemia and hereditary persistence of fetal hemoglobin in the Chinese Zhuang population.

Journal: Journal of clinical laboratory analysis

Published: May 09, 2017

Complex Interaction of Hb Q-Thailand with α0- and β0-Thalassemia in a Chinese Family.

Complex Interaction of Hb Q-Thailand with α0- and β0-Thalassemia in a Chinese Family.

Journal: Hemoglobin

Published: May 06, 2017

A Novel Mutation of the α2-Globin Gene Causing α+-Thalassemia: Hb Nanning (HBA2: c.369_370delinsGA).

A Novel Mutation of the α2-Globin Gene Causing α+-Thalassemia: Hb Nanning (HBA2: c.369_370delinsGA).

Journal: Hemoglobin

Published: April 12, 2017

Common genetic polymorphisms at three loci affect HbF levels in β-thalassemia patients from Southern China.

Common genetic polymorphisms at three loci affect HbF levels in β-thalassemia patients from Southern China.

Journal: Blood cells, molecules & diseases

Published: August 08, 2016

Identification of a Novel β-Globin Mutation (HBB: C.189_195delTCATGGC) in a Chinese Family.

Identification of a Novel β-Globin Mutation (HBB: C.189_195delTCATGGC) in a Chinese Family.

Journal: Hemoglobin

Published: August 06, 2016

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Last Updated: 09/26/2026