Lia M. Bevilaqua
Natal, RN 59078900, Brazil
Last Updated: 09/26/2026
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Found 11 publications
NMDARs control object recognition memory destabilization and reconsolidation.
Journal: Brain research bulletin
Published: December 23, 2022
On the effect of hippocampal c-Jun N-terminal kinase inhibition on object recognition memory.
Journal: Frontiers in behavioral neuroscience
Published: September 23, 2022
Avoidance memory requires CaMKII activity to persist after recall.
Journal: Molecular brain
Published: September 01, 2021
Reactivation-dependent amnesia for object recognition memory is contingent on hippocampal theta-gamma coupling during recall.
Journal: Learning & memory (Cold Spring Harbor, N.Y.)
Published: August 06, 2021
Dopamine controls whether new declarative information updates reactivated memories through reconsolidation.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: July 13, 2021
GluN2B and GluN2A-containing NMDAR are differentially involved in extinction memory destabilization and restabilization during reconsolidation.
Journal: Scientific reports
Published: September 25, 2020
mTOR inhibition impairs extinction memory reconsolidation.
Journal: Learning & memory (Cold Spring Harbor, N.Y.)
Published: June 01, 2020
PERK, mTORC1 and eEF2 interplay during long term potentiation: An Editorial for 'Genetic removal of eIF2a kinase PERK in mice enables hippocampal L-LTP independent of mTORC1 activity' on page 133.
Journal: Journal of neurochemistry
Published: May 18, 2018
Inactivation of the dorsal hippocampus or the medial prefrontal cortex impairs retrieval but has differential effect on spatial memory reconsolidation.
Journal: Neurobiology of learning and memory
Published: October 23, 2014
State-dependent effect of dopamine D₁/D₅ receptors inactivation on memory destabilization and reconsolidation.
Journal: Behavioural brain research
Published: May 20, 2014
Showing 1-11 of 11
Last Updated: 09/26/2026