Lian-kun Song

State Key Laboratory Of Bioactive Substances And Functions Of Natural Medicines, Chinese Academy Of Medical Sciences And Peking Union Medical College
Beijing, 100050, China

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 9 publications

Corrigendum to 'Deletion in exon 5 of the SNCA gene and exposure to rotenone leads to oligomerization of α-synuclein and toxicity to PC12 cells' [Brain Research Bulletin 90 (2013) 127-131].

Corrigendum to 'Deletion in exon 5 of the SNCA gene and exposure to rotenone leads to oligomerization of α-synuclein and toxicity to PC12 cells' [Brain Research Bulletin 90 (2013) 127-131].

Journal: Brain research bulletin

Published: May 20, 2026

Corrigendum to "Over-expression of α-synuclein 98 triggers intracellular oxidative stress and enhances susceptibility to rotenone" [Neurosci. Lett. 491 (2011) 148-152].

Corrigendum to "Over-expression of α-synuclein 98 triggers intracellular oxidative stress and enhances susceptibility to rotenone" [Neurosci. Lett. 491 (2011) 148-152].

Journal: Neuroscience letters

Published: April 22, 2025

Endoplasmic reticulum stress, an important factor in the development of Parkinson's disease.

Endoplasmic reticulum stress, an important factor in the development of Parkinson's disease.

Journal: Toxicology letters

Published: October 08, 2019

A new coumarin derivative, IMM-H004, attenuates okadaic acid-induced spatial memory impairment in rats.

A new coumarin derivative, IMM-H004, attenuates okadaic acid-induced spatial memory impairment in rats.

Journal: Acta pharmacologica Sinica

Published: September 01, 2015

Environment-contact administration of rotenone: A new rodent model of Parkinson's disease.

Environment-contact administration of rotenone: A new rodent model of Parkinson's disease.

Journal: Behavioural brain research

Published: April 03, 2015

Targeted Overexpression of α-Synuclein by rAAV2/1 Vectors Induces Progressive Nigrostriatal Degeneration and Increases Vulnerability to MPTP in Mouse.

Targeted Overexpression of α-Synuclein by rAAV2/1 Vectors Induces Progressive Nigrostriatal Degeneration and Increases Vulnerability to MPTP in Mouse.

Journal: PloS one

Published: December 04, 2014

IMM-H004, a novel coumarin derivative compound, attenuates the production of inflammatory mediatory mediators in lipopolysaccharide-activated BV2 microglia.

IMM-H004, a novel coumarin derivative compound, attenuates the production of inflammatory mediatory mediators in lipopolysaccharide-activated BV2 microglia.

Journal: Brain research bulletin

Published: April 24, 2014

The nuclear accumulation of alpha-synuclein is mediated by importin alpha and promotes neurotoxicity by accelerating the cell cycle.

The nuclear accumulation of alpha-synuclein is mediated by importin alpha and promotes neurotoxicity by accelerating the cell cycle.

Journal: Neuropharmacology

Published: April 12, 2013

Deletion in exon 5 of the SNCA gene and exposure to rotenone leads to oligomerization of α-synuclein and toxicity to PC12 cells.

Deletion in exon 5 of the SNCA gene and exposure to rotenone leads to oligomerization of α-synuclein and toxicity to PC12 cells.

Journal: Brain research bulletin

Published: August 06, 2012

Showing 1-9 of 9

Last Updated: 09/26/2026