Lihong Lu

Zhongshan, China

Last Updated: 09/26/2026

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Found 12 publications

Sirt7/HIC1 complex participates in hyperglycaemia-mediated EndMT via modulation of SDC1 expression in diabetic kidney disease and metabolic memory.

Sirt7/HIC1 complex participates in hyperglycaemia-mediated EndMT via modulation of SDC1 expression in diabetic kidney disease and metabolic memory.

Journal: Journal of cellular and molecular medicine

Published: September 17, 2023

HSK3486 Inhibits Colorectal Cancer Growth by Promoting Oxidative Stress and ATPase Inhibitory Factor 1 Activation.

HSK3486 Inhibits Colorectal Cancer Growth by Promoting Oxidative Stress and ATPase Inhibitory Factor 1 Activation.

Journal: Digestive diseases and sciences

Published: June 08, 2023

Fasting-Mimicking Diet Drives Antitumor Immunity against Colorectal Cancer by Reducing IgA-Producing Cells.

Fasting-Mimicking Diet Drives Antitumor Immunity against Colorectal Cancer by Reducing IgA-Producing Cells.

Journal: Cancer research

Published: January 31, 2023

Propofol protects against high glucose-mediated endothelial injury via inhibition of COX2 and iNOS expressions.

Propofol protects against high glucose-mediated endothelial injury via inhibition of COX2 and iNOS expressions.

Journal: Acta biochimica et biophysica Sinica

Published: May 24, 2022

Epigenetics in the pathogenesis of diabetic nephropathy.

Epigenetics in the pathogenesis of diabetic nephropathy.

Journal: Acta biochimica et biophysica Sinica

Published: February 07, 2022

Sirt7 associates with ELK1 to participate in hyperglycemia memory and diabetic nephropathy via modulation of DAPK3 expression and endothelial inflammation.

Sirt7 associates with ELK1 to participate in hyperglycemia memory and diabetic nephropathy via modulation of DAPK3 expression and endothelial inflammation.

Journal: Translational research : the journal of laboratory and clinical medicine

Published: December 31, 2021

The SETD8/ELK1/bach1 complex regulates hyperglycaemia-mediated EndMT in diabetic nephropathy.

The SETD8/ELK1/bach1 complex regulates hyperglycaemia-mediated EndMT in diabetic nephropathy.

Journal: Journal of translational medicine

Published: December 17, 2021

SETD8 cooperates with MZF1 to participate in hyperglycemia-induced endothelial inflammation via elevation of WNT5A levels in diabetic nephropathy.

SETD8 cooperates with MZF1 to participate in hyperglycemia-induced endothelial inflammation via elevation of WNT5A levels in diabetic nephropathy.

Journal: Cellular & molecular biology letters

Published: December 01, 2021

c-Myc participates in high glucose-mediated endothelial inflammation via upregulation of IRAK1 expression in diabetic nephropathy.

c-Myc participates in high glucose-mediated endothelial inflammation via upregulation of IRAK1 expression in diabetic nephropathy.

Journal: Cellular signalling

Published: September 15, 2021

KMT5A downregulation participated in High Glucose-mediated EndMT via Upregulation of ENO1 Expression in Diabetic Nephropathy.

KMT5A downregulation participated in High Glucose-mediated EndMT via Upregulation of ENO1 Expression in Diabetic Nephropathy.

Journal: International journal of biological sciences

Published: May 18, 2021

ets1 associates with KMT5A to participate in high glucose-mediated EndMT via upregulation of PFN2 expression in diabetic nephropathy.

ets1 associates with KMT5A to participate in high glucose-mediated EndMT via upregulation of PFN2 expression in diabetic nephropathy.

Journal: Molecular medicine (Cambridge, Mass.)

Published: February 16, 2021

The CREB/KMT5A complex regulates PTP1B to modulate high glucose-induced endothelial inflammatory factor levels in diabetic nephropathy.

The CREB/KMT5A complex regulates PTP1B to modulate high glucose-induced endothelial inflammatory factor levels in diabetic nephropathy.

Journal: Cell death & disease

Published: January 13, 2021

Showing 1-12 of 12

Last Updated: 09/26/2026