Lisa A. Kohn

Allergy and Immunology | Pediatrics | Pediatric Allergy and Immunology

2020 Santa Monica Blvd
Suite 210
Santa Monica, CA 90404

Last Updated: 09/26/2026

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Found 14 publications

UTX coordinates TCF1 and STAT3 to control progenitor CD8+ T cell fate in autoimmune diabetes.

UTX coordinates TCF1 and STAT3 to control progenitor CD8+ T cell fate in autoimmune diabetes.

Journal: The Journal of clinical investigation

Published: December 16, 2025

UTX coordinates TCF1 and STAT3 to control progenitor CD8+ T cell fate in autoimmune diabetes.

UTX coordinates TCF1 and STAT3 to control progenitor CD8+ T cell fate in autoimmune diabetes.

Journal: The Journal of clinical investigation

Published: June 03, 2025

UTX Coordinates TCF1 and STAT3 to Control Progenitor CD8+ T cell Fate in Autoimmune Diabetes.

UTX Coordinates TCF1 and STAT3 to Control Progenitor CD8+ T cell Fate in Autoimmune Diabetes.

Journal: bioRxiv : the preprint server for biology

Published: January 07, 2025

Electronic health record signatures identify undiagnosed patients with common variable immunodeficiency disease.

Electronic health record signatures identify undiagnosed patients with common variable immunodeficiency disease.

Journal: Science translational medicine

Published: May 01, 2024

Immunotherapy responsive neuroinflammation in a child with FAS-associated death-domain mutation.

Immunotherapy responsive neuroinflammation in a child with FAS-associated death-domain mutation.

Journal: Clinical genetics

Published: June 09, 2022

Gene Therapies for Primary Immune Deficiencies.

Gene Therapies for Primary Immune Deficiencies.

Journal: Frontiers in immunology

Published: January 03, 2021

Commercial immunoglobulin products contain cross-reactive but not neutralizing antibodies against SARS-CoV-2.

Commercial immunoglobulin products contain cross-reactive but not neutralizing antibodies against SARS-CoV-2.

Journal: The Journal of allergy and clinical immunology

Published: October 04, 2020

Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome.

Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome.

Journal: The Journal of experimental medicine

Published: June 10, 2020

Novel Compound Heterozygote Variations in FADD Identified to Cause FAS-Associated Protein with Death Domain Deficiency.

Novel Compound Heterozygote Variations in FADD Identified to Cause FAS-Associated Protein with Death Domain Deficiency.

Journal: Journal of clinical immunology

Published: November 29, 2019

Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome.

Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome.

Journal: The Journal of experimental medicine

Published: September 25, 2019

Treatment of systemic mastocytosis in an infant with midostaurin.

Treatment of systemic mastocytosis in an infant with midostaurin.

Journal: The journal of allergy and clinical immunology. In practice

Published: March 06, 2019

Human lymphoid development in the absence of common γ-chain receptor signaling.

Human lymphoid development in the absence of common γ-chain receptor signaling.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: April 29, 2014

Showing 1-12 of 14

Last Updated: 09/26/2026