Long-fei Luo
Wuhan, China
Last Updated: 09/26/2026
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Found 10 publications
Psychological stress exacerbates vitiligo via dysregulated proteolysis of substance P and MRGPRX2-dependent mast cell activation.
Journal: Journal of dermatological science
Published: January 08, 2026
Oxidative stress impairs the expansion of regulatory T cells in active vitiligo via dysregulated CGRP-RAMP1-Gαi3 signaling.
Journal: Free radical biology & medicine
Published: November 03, 2025
Increased splicing of CXCR3 isoform B (CXCR3B) by impaired NRF2 signaling leads to melanocyte apoptosis in active vitiligo.
Journal: Free radical biology & medicine
Published: September 15, 2024
Acceleration of melanocyte senescence by the proinflammatory cytokines IFNγ and TNFα impairs the repigmentation response of vitiligo patients to narrowband ultraviolet B (NBUVB) phototherapy.
Journal: Mechanisms of ageing and development
Published: November 30, 2022
Uncoupling melanogenesis from proliferation in epidermal melanocytes responding to stimulation with psoriasis-related proinflammatory cytokines.
Journal: Journal of dermatological science
Published: April 27, 2022
LINC00937 suppresses keloid fibroblast proliferation and extracellular matrix deposition by targeting the miR-28-5p/MC1R axis.
Journal: Histology and histopathology
Published: September 06, 2021
Intramelanocytic Acidification Plays a Role in the Antimelanogenic and Antioxidative Properties of Vitamin C and Its Derivatives.
Journal: Oxidative medicine and cellular longevity
Published: December 30, 2018
Asymmetric stem-cell division ensures sustained keratinocyte hyperproliferation in psoriatic skin lesions.
Journal: International journal of molecular medicine
Published: July 05, 2015
Insufficient expression of the melanocortin-1 receptor by human dermal fibroblasts contributes to excess collagen synthesis in keloid scars.
Journal: Experimental dermatology
Published: September 17, 2013
Showing 1-10 of 10
Last Updated: 09/26/2026