Luca Goitre

Orbassano, 10043, Italy

Last Updated: 09/26/2026

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Found 17 publications

Heterozygous Loss of KRIT1 in Mice Affects Metabolic Functions of the Liver, Promoting Hepatic Oxidative and Glycative Stress.

Heterozygous Loss of KRIT1 in Mice Affects Metabolic Functions of the Liver, Promoting Hepatic Oxidative and Glycative Stress.

Journal: International journal of molecular sciences

Published: August 12, 2022

Production of KRIT1-knockout and KRIT1-knockin Mouse Embryonic Fibroblasts as Cellular Models of CCM Disease.

Production of KRIT1-knockout and KRIT1-knockin Mouse Embryonic Fibroblasts as Cellular Models of CCM Disease.

Journal: Methods in molecular biology (Clifton, N.J.)

Published: June 12, 2020

Biological Activities, Health Benefits, and Therapeutic Properties of Avenanthramides: From Skin Protection to Prevention and Treatment of Cerebrovascular Diseases.

Biological Activities, Health Benefits, and Therapeutic Properties of Avenanthramides: From Skin Protection to Prevention and Treatment of Cerebrovascular Diseases.

Journal: Oxidative medicine and cellular longevity

Published: April 27, 2018

KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease.

KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease.

Journal: Free radical biology & medicine

Published: March 26, 2017

Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium.

Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium.

Journal: Scientific reports

Published: March 13, 2017

Beyond multiple mechanisms and a unique drug: Defective autophagy as pivotal player in cerebral cavernous malformation pathogenesis and implications for targeted therapies.

Beyond multiple mechanisms and a unique drug: Defective autophagy as pivotal player in cerebral cavernous malformation pathogenesis and implications for targeted therapies.

Journal: Rare diseases (Austin, Tex.)

Published: November 17, 2015

Cytochrome P450 and matrix metalloproteinase genetic modifiers of disease severity in Cerebral Cavernous Malformation type 1.

Cytochrome P450 and matrix metalloproteinase genetic modifiers of disease severity in Cerebral Cavernous Malformation type 1.

Journal: Free radical biology & medicine

Published: November 12, 2015

Defective autophagy is a key feature of cerebral cavernous malformations.

Defective autophagy is a key feature of cerebral cavernous malformations.

Journal: EMBO molecular medicine

Published: September 30, 2015

Strategy for identifying repurposed drugs for the treatment of cerebral cavernous malformation.

Strategy for identifying repurposed drugs for the treatment of cerebral cavernous malformation.

Journal: Circulation

Published: December 10, 2014

Evaluation of the bioactive properties of avenanthramide analogs produced in recombinant yeast.

Evaluation of the bioactive properties of avenanthramide analogs produced in recombinant yeast.

Journal: BioFactors (Oxford, England)

Published: October 09, 2014

The Ras superfamily of small GTPases: the unlocked secrets.

The Ras superfamily of small GTPases: the unlocked secrets.

Journal: Methods in molecular biology (Clifton, N.J.)

Published: January 29, 2014

Fluorescence microscopy study of Rap1 subcellular localization.

Fluorescence microscopy study of Rap1 subcellular localization.

Journal: Methods in molecular biology (Clifton, N.J.)

Published: January 29, 2014

Showing 1-12 of 17

Last Updated: 09/26/2026