Lukas Alan
Prague, PR, Czech Republic
Last Updated: 09/26/2026
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Found 26 publications
Chronic hypoxia protects the mouse heart from oxidative stress via HIF-1α-mitochondria crosstalk.
Journal: Cellular and molecular life sciences : CMLS
Published: January 21, 2026
The cytochrome c oxidase subunit COX6B1 is required for redox-sensitive early assembly and late stabilization of complex IV.
Journal: The Journal of biological chemistry
Published: July 28, 2025
OPA1 Deficiency Impairs NGF Signaling and Drives Sympathetic Neurodegeneration.
Journal: JACC. Basic to translational science
Published: July 16, 2025
Mitochondrial metabolism and hypoxic signaling in differentiated human cardiomyocyte AC16 cell line.
Mitochondrial metabolism and hypoxic signaling in differentiated human cardiomyocyte AC16 cell line.
Journal: American journal of physiology. Cell physiology
Published: April 17, 2025
Cardioprotective Effect of Chronic Hypoxia Involves Inhibition of Mitochondrial Permeability Transition Pore Opening.
Journal: Physiological research
Published: November 19, 2024
Opantimirs: A class of antagonizing microRNAs that upregulate Opa1 and improve mitochondrial and disuse myopathies.
Journal: Cell reports. Medicine
Published: November 13, 2024
HIF-1α limits myocardial infarction by promoting mitophagy in mouse hearts adapted to chronic hypoxia.
Journal: Acta physiologica (Oxford, England)
Published: October 30, 2023
Oxidization of optic atrophy 1 cysteines occurs during heart ischemia-reperfusion and amplifies cell death by oxidative stress.
Journal: Redox biology
Published: March 24, 2023
Antioxidant Role and Cardiolipin Remodeling by Redox-Activated Mitochondrial Ca2+-Independent Phospholipase A2γ in the Brain.
Journal: Antioxidants (Basel, Switzerland)
Published: December 28, 2021
Last Updated: 09/26/2026