Lulu Bai

Chongqing, China

Last Updated: 09/26/2026

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Found 10 publications

Exposure to carbon black nanoparticles during pregnancy aggravates lipopolysaccharide-induced lung injury in offspring: an intergenerational effect.

Exposure to carbon black nanoparticles during pregnancy aggravates lipopolysaccharide-induced lung injury in offspring: an intergenerational effect.

Journal: American journal of physiology. Lung cellular and molecular physiology

Published: September 29, 2021

Pregnancy exposure to carbon black nanoparticles induced neurobehavioral deficits that are associated with altered m6A modification in offspring.

Pregnancy exposure to carbon black nanoparticles induced neurobehavioral deficits that are associated with altered m6A modification in offspring.

Journal: Neurotoxicology

Published: October 11, 2019

Gut-brain communication in hyperfunction of 5-hydroxytryptamine induced by oral zinc oxide nanoparticles exposure in young mice.

Gut-brain communication in hyperfunction of 5-hydroxytryptamine induced by oral zinc oxide nanoparticles exposure in young mice.

Journal: Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association

Published: August 20, 2019

Synaptic dopamine release is positively regulated by SNAP-25 that involves in benzo[a]pyrene-induced neurotoxicity.

Synaptic dopamine release is positively regulated by SNAP-25 that involves in benzo[a]pyrene-induced neurotoxicity.

Journal: Chemosphere

Published: March 05, 2019

Exposure to carbon black nanoparticles during pregnancy persistently damages the cerebrovascular function in female mice.

Exposure to carbon black nanoparticles during pregnancy persistently damages the cerebrovascular function in female mice.

Journal: Toxicology

Published: February 01, 2019

Autophagy-dependent release of zinc ions is critical for acute lung injury triggered by zinc oxide nanoparticles.

Autophagy-dependent release of zinc ions is critical for acute lung injury triggered by zinc oxide nanoparticles.

Journal: Nanotoxicology

Published: October 16, 2018

Maternal exposure to traffic pollutant causes impairment of spermatogenesis and alterations of genome-wide mRNA and microRNA expression in F2 male mice.

Maternal exposure to traffic pollutant causes impairment of spermatogenesis and alterations of genome-wide mRNA and microRNA expression in F2 male mice.

Journal: Environmental toxicology and pharmacology

Published: July 14, 2018

m6A Demethylase FTO Regulates Dopaminergic Neurotransmission Deficits Caused by Arsenite.

m6A Demethylase FTO Regulates Dopaminergic Neurotransmission Deficits Caused by Arsenite.

Journal: Toxicological sciences : an official journal of the Society of Toxicology

Published: July 09, 2018

Ferroptosis is newly characterized form of neuronal cell death in response to arsenite exposure.

Ferroptosis is newly characterized form of neuronal cell death in response to arsenite exposure.

Journal: Neurotoxicology

Published: November 17, 2017

Inhibition of α-Synuclein contributes to the ameliorative effects of dietary flavonoids luteolin on arsenite-induced apoptotic cell death in the dopaminergic PC12 cells.

Inhibition of α-Synuclein contributes to the ameliorative effects of dietary flavonoids luteolin on arsenite-induced apoptotic cell death in the dopaminergic PC12 cells.

Journal: Toxicology mechanisms and methods

Published: June 07, 2017

Showing 1-10 of 10

Last Updated: 09/26/2026