Lynda Zeboudj
Paris, 75005, France
Last Updated: 09/26/2026
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Found 21 publications
Paclitaxel induced-mechanical hypersensitivity is associated with time-dependant pro-inflammatory galectin-3+ macrophages accumulation in the dorsal root ganglia.
Journal: Neurobiology of pain (Cambridge, Mass.)
Published: April 02, 2026
Microglial Activation in Nociplastic Pain: From Preclinical Models to PET Neuroimaging and Implications for Targeted Therapeutic Strategies.
Journal: International journal of molecular sciences
Published: November 13, 2025
Macrophage to neuron communication via extracellular vesicles in neuropathic pain conditions.
Journal: Heliyon
Published: August 27, 2024
Galectin-3 activates spinal microglia to induce inflammatory nociception in wild type but not in mice modelling Alzheimer's disease.
Journal: Nature communications
Published: August 31, 2022
CD206+/MHCII- macrophage accumulation at nerve injury site correlates with attenuation of allodynia in TASTPM mouse model of Alzheimer's disease.
Journal: Brain, behavior, & immunity - health
Published: August 26, 2022
Silencing miR-21-5p in sensory neurons reverses neuropathic allodynia via activation of TGF-β-related pathway in macrophages.
Journal: The Journal of clinical investigation
Published: August 12, 2022
Dorsal root ganglia CX3CR1 expressing monocytes/macrophages contribute to arthritis pain.
Journal: Brain, behavior, and immunity
Published: March 29, 2022
Inhibition of Bruton's TK regulates macrophage NF-κB and NLRP3 inflammasome activation in metabolic inflammation.
Journal: British journal of pharmacology
Published: January 21, 2020
A Biased Agonist at Immunometabolic Receptor GPR84 Causes Distinct Functional Effects in Macrophages.
Journal: ACS chemical biology
Published: August 30, 2019
Efferocytosis perpetuates substance accumulation inside macrophage populations.
Journal: Proceedings. Biological sciences
Published: June 05, 2019
EGF receptor blockade in immune cells reduces experimental atherosclerosis
Journal: Medecine sciences : M/S
Published: August 02, 2018
Smooth muscle cells-derived CXCL10 prevents endothelial healing through PI3Kγ-dependent T cells response.
Journal: Cardiovascular research
Published: July 27, 2018
Last Updated: 09/26/2026