Madeleine C. Mcneill
Bristol, ENG BS28HW, United Kingdom
Last Updated: 09/26/2026
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Found 8 publications
Extracellular matrix stiffness primes the NLRP3-Inflammasome by promoting NLRP3 gene expression via FAK/mTOR/nuclear-actin/SREBP signalling in vascular smooth muscle cells.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: March 16, 2026
Extracellular matrix stiffness controls cardiac fibroblast proliferation via the nuclear factor-Y (NF-Y) transcription factor.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: August 31, 2023
Substrate stiffness promotes vascular smooth muscle cell calcification by reducing the levels of nuclear actin monomers.
Journal: Journal of molecular and cellular cardiology
Published: July 27, 2023
Combined role for YAP-TEAD and YAP-RUNX2 signalling in substrate-stiffness regulation of cardiac fibroblast proliferation.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: March 11, 2022
Cyclic-AMP Increases Nuclear Actin Monomer Which Promotes Proteasomal Degradation of RelA/p65 Leading to Anti-Inflammatory Effects.
Journal: Cells
Published: January 18, 2022
Nuclear actin regulates cell proliferation and migration via inhibition of SRF and TEAD.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: November 14, 2019
Elevated cyclic-AMP represses expression of exchange protein activated by cAMP (EPAC1) by inhibiting YAP-TEAD activity and HDAC-mediated histone deacetylation.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: March 06, 2019
Antiproliferative and Antimigratory Effects of a Novel YAP-TEAD Interaction Inhibitor Identified Using in Silico Molecular Docking.
Journal: Journal of medicinal chemistry
Published: January 15, 2019
Showing 1-8 of 8
Last Updated: 09/26/2026