Madeleine E. Cunningham

Glasgow, SCT, United Kingdom

Last Updated: 09/26/2026

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Found 15 publications

Correction: CCR1 and CCR2 Coexpression on Monocytes Is Nonredundant and Delineates a Distinct Monocyte Subpopulation.

Correction: CCR1 and CCR2 Coexpression on Monocytes Is Nonredundant and Delineates a Distinct Monocyte Subpopulation.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: October 09, 2024

CCR1 and CCR2 Coexpression on Monocytes Is Nonredundant and Delineates a Distinct Monocyte Subpopulation.

CCR1 and CCR2 Coexpression on Monocytes Is Nonredundant and Delineates a Distinct Monocyte Subpopulation.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: January 03, 2024

Axolemmal nanoruptures arising from paranodal membrane injury induce secondary axon degeneration in murine Guillain-Barré syndrome.

Axolemmal nanoruptures arising from paranodal membrane injury induce secondary axon degeneration in murine Guillain-Barré syndrome.

Journal: Journal of the peripheral nervous system : JPNS

Published: December 09, 2022

The endogenous calpain inhibitor calpastatin attenuates axon degeneration in murine Guillain-Barré syndrome.

The endogenous calpain inhibitor calpastatin attenuates axon degeneration in murine Guillain-Barré syndrome.

Journal: Journal of the peripheral nervous system : JPNS

Published: September 27, 2022

Complement inhibition prevents glial nodal membrane injury in a GM1 antibody-mediated mouse model.

Complement inhibition prevents glial nodal membrane injury in a GM1 antibody-mediated mouse model.

Journal: Brain communications

Published: July 05, 2022

Real time imaging of intra-axonal calcium flux in an explant mouse model of axonal Guillain-Barré syndrome.

Real time imaging of intra-axonal calcium flux in an explant mouse model of axonal Guillain-Barré syndrome.

Journal: Experimental neurology

Published: January 20, 2022

Schwann cell nodal membrane disruption triggers bystander axonal degeneration in a Guillain-Barré syndrome mouse model.

Schwann cell nodal membrane disruption triggers bystander axonal degeneration in a Guillain-Barré syndrome mouse model.

Journal: The Journal of clinical investigation

Published: January 14, 2022

Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain-Barré syndrome.

Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain-Barré syndrome.

Journal: Journal of the peripheral nervous system : JPNS

Published: January 28, 2020

Glial Sulfatides and Neuronal Complex Gangliosides Are Functionally Interdependent in Maintaining Myelinating Axon Integrity.

Glial Sulfatides and Neuronal Complex Gangliosides Are Functionally Interdependent in Maintaining Myelinating Axon Integrity.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience

Published: August 14, 2018

Differential binding patterns of anti-sulfatide antibodies to glial membranes.

Differential binding patterns of anti-sulfatide antibodies to glial membranes.

Journal: Journal of neuroimmunology

Published: May 31, 2018

Zika virus tropism and interactions in myelinating neural cell cultures: CNS cells and myelin are preferentially affected.

Zika virus tropism and interactions in myelinating neural cell cultures: CNS cells and myelin are preferentially affected.

Journal: Acta neuropathologica communications

Published: May 31, 2017

C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy.

C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy.

Journal: Acta neuropathologica communications

Published: February 17, 2016

Showing 1-12 of 15

Last Updated: 09/26/2026