Madeline Murphy

Dublin, L, Ireland

Last Updated: 09/26/2026

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Found 14 publications

Bone morphogenetic protein-3 is a negative regulator of transforming growth factor beta and fibrosis.

Bone morphogenetic protein-3 is a negative regulator of transforming growth factor beta and fibrosis.

Journal: Biochemical and biophysical research communications

Published: July 12, 2024

Post-intubation subglottic stenosis: aetiology at the cellular and molecular level.

Post-intubation subglottic stenosis: aetiology at the cellular and molecular level.

Journal: European respiratory review : an official journal of the European Respiratory Society

Published: July 06, 2020

Frequency, impact and a preclinical study of novel ERBB gene family mutations in HER2-positive breast cancer.

Frequency, impact and a preclinical study of novel ERBB gene family mutations in HER2-positive breast cancer.

Journal: Therapeutic advances in medical oncology

Published: November 19, 2017

Profibrotic IHG-1 complexes with renal disease associated HSPA5 and TRAP1 in mitochondria.

Profibrotic IHG-1 complexes with renal disease associated HSPA5 and TRAP1 in mitochondria.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: October 06, 2016

A Friend in Need: Activated Protein C Stabilizes YB-1 during Renal Ischemia Reperfusion Injury.

A Friend in Need: Activated Protein C Stabilizes YB-1 during Renal Ischemia Reperfusion Injury.

Journal: Journal of the American Society of Nephrology : JASN

Published: May 28, 2015

Overexpression of the microRNA miR-433 promotes resistance to paclitaxel through the induction of cellular senescence in ovarian cancer cells.

Overexpression of the microRNA miR-433 promotes resistance to paclitaxel through the induction of cellular senescence in ovarian cancer cells.

Journal: Cancer medicine

Published: September 17, 2014

Targeting cellular drivers and counter-regulators of hyperglycaemia- and transforming growth factor-β1-associated profibrotic responses in diabetic kidney disease.

Targeting cellular drivers and counter-regulators of hyperglycaemia- and transforming growth factor-β1-associated profibrotic responses in diabetic kidney disease.

Journal: Experimental physiology

Published: August 03, 2014

IHG-1 increases mitochondrial fusion and bioenergetic function.

IHG-1 increases mitochondrial fusion and bioenergetic function.

Journal: Diabetes

Published: July 11, 2014

Epigenetic unsilencing reverses renal fibrosis.

Epigenetic unsilencing reverses renal fibrosis.

Journal: Journal of the American Society of Nephrology : JASN

Published: February 01, 2014

Lipoxins attenuate renal fibrosis by inducing let-7c and suppressing TGFβR1.

Lipoxins attenuate renal fibrosis by inducing let-7c and suppressing TGFβR1.

Journal: Journal of the American Society of Nephrology : JASN

Published: March 23, 2013

IHG-1 amplifies TGF-β1 signalling and mitochondrial biogenesis and is increased in diabetic kidney disease.

IHG-1 amplifies TGF-β1 signalling and mitochondrial biogenesis and is increased in diabetic kidney disease.

Journal: Current opinion in nephrology and hypertension

Published: November 21, 2012

IHG-1 must be localised to mitochondria to decrease Smad7 expression and amplify TGF-β1-induced fibrotic responses.

IHG-1 must be localised to mitochondria to decrease Smad7 expression and amplify TGF-β1-induced fibrotic responses.

Journal: Biochimica et biophysica acta

Published: August 07, 2012

Showing 1-12 of 14

Last Updated: 09/26/2026