Madhura Panse

Otfried Mueller Street 10
Tuebingen, BW 72076, Germany

Last Updated: 09/26/2026

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Found 9 publications

Glucose Responsiveness of β-Cells Depends on Fatty Acids.

Glucose Responsiveness of β-Cells Depends on Fatty Acids.

Journal: Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association

Published: April 16, 2019

The Expression of Aldolase B in Islets Is Negatively Associated With Insulin Secretion in Humans.

The Expression of Aldolase B in Islets Is Negatively Associated With Insulin Secretion in Humans.

Journal: The Journal of clinical endocrinology and metabolism

Published: April 12, 2018

Palmitate and insulin counteract glucose-induced thioredoxin interacting protein (TXNIP) expression in insulin secreting cells via distinct mechanisms.

Palmitate and insulin counteract glucose-induced thioredoxin interacting protein (TXNIP) expression in insulin secreting cells via distinct mechanisms.

Journal: PloS one

Published: December 04, 2017

Point mutation of Ffar1 abrogates fatty acid-dependent insulin secretion, but protects against HFD-induced glucose intolerance.

Point mutation of Ffar1 abrogates fatty acid-dependent insulin secretion, but protects against HFD-induced glucose intolerance.

Journal: Molecular metabolism

Published: November 16, 2016

Metabolic crosstalk between fatty pancreas and fatty liver: effects on local inflammation and insulin secretion.

Metabolic crosstalk between fatty pancreas and fatty liver: effects on local inflammation and insulin secretion.

Journal: Diabetologia

Published: September 29, 2016

Protein kinase Cδ regulates nuclear export of FOXO1 through phosphorylation of the chaperone 14-3-3ζ.

Protein kinase Cδ regulates nuclear export of FOXO1 through phosphorylation of the chaperone 14-3-3ζ.

Journal: Diabetologia

Published: April 27, 2015

Mice Lacking Free Fatty Acid Receptor 1 (GPR40/FFAR1) are Protected Against Conjugated Linoleic Acid-Induced Fatty Liver but Develop Inflammation and Insulin Resistance in the Brain.

Mice Lacking Free Fatty Acid Receptor 1 (GPR40/FFAR1) are Protected Against Conjugated Linoleic Acid-Induced Fatty Liver but Develop Inflammation and Insulin Resistance in the Brain.

Journal: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology

Published: January 20, 2015

Activation of extracellular signal-regulated protein kinases 1 and 2 (ERK1/2) by free fatty acid receptor 1 (FFAR1/GPR40) protects from palmitate-induced beta cell death, but plays no role in insulin secretion.

Activation of extracellular signal-regulated protein kinases 1 and 2 (ERK1/2) by free fatty acid receptor 1 (FFAR1/GPR40) protects from palmitate-induced beta cell death, but plays no role in insulin secretion.

Journal: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology

Published: January 15, 2015

Detection of free fatty acid receptor 1 expression: the critical role of negative and positive controls.

Detection of free fatty acid receptor 1 expression: the critical role of negative and positive controls.

Journal: Diabetologia

Published: October 14, 2013

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Last Updated: 09/26/2026