Magdalena S. Mroz
Dublin, L, Ireland
Last Updated: 09/26/2026
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Found 9 publications
The secondary bile acid, lithocholic acid, inhibits cystic fibrosis transmembrane conductance regulator expression and activity in colonic epithelial cells.
Journal: American journal of physiology. Gastrointestinal and liver physiology
Published: December 26, 2025
Ursodeoxycholic acid inhibits ENaC and Na/K pump activity to restore airway surface liquid height in cystic fibrosis bronchial epithelial cells.
Journal: Steroids
Published: May 25, 2019
The bile acids, deoxycholic acid and ursodeoxycholic acid, regulate colonic epithelial wound healing.
Journal: American journal of physiology. Gastrointestinal and liver physiology
Published: January 20, 2018
Targeting the PI3K/Akt/mTOR signalling pathway in Cystic Fibrosis.
Journal: Scientific reports
Published: February 06, 2017
Bile acids stimulate chloride secretion through CFTR and calcium-activated Cl- channels in Calu-3 airway epithelial cells.
Journal: American journal of physiology. Lung cellular and molecular physiology
Published: July 05, 2014
Ursodeoxycholic acid attenuates colonic epithelial secretory function.
Journal: The Journal of physiology
Published: March 20, 2013
The bile acid receptor, TGR5, regulates basal and cholinergic-induced secretory responses in rat colon.
Journal: Neurogastroenterology and motility
Published: February 15, 2013
Epidermal growth factor chronically upregulates Ca(2+)-dependent Cl(-) conductance and TMEM16A expression in intestinal epithelial cells.
Journal: The Journal of physiology
Published: February 22, 2012
Showing 1-9 of 9
Last Updated: 09/26/2026