Maike Rubsamen-Hartlage

Leipzig, SN 04103, Germany

Last Updated: 09/26/2026

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Found 21 publications

Monomers, Dimers, and Oligomers of Pyroglutamate-Modified α-Synuclein Fragments Exhibit Distinct Biophysical Characteristics.

Monomers, Dimers, and Oligomers of Pyroglutamate-Modified α-Synuclein Fragments Exhibit Distinct Biophysical Characteristics.

Journal: ACS chemical neuroscience

Published: April 30, 2025

Identification of isoAsp7-Aβ as a major Aβ variant in Alzheimer's disease, dementia with Lewy bodies and vascular dementia.

Identification of isoAsp7-Aβ as a major Aβ variant in Alzheimer's disease, dementia with Lewy bodies and vascular dementia.

Journal: Acta neuropathologica

Published: August 27, 2024

Young human alpha synuclein transgenic (BAC-SNCA) mice display sex- and gene-dose-dependent phenotypic disturbances.

Young human alpha synuclein transgenic (BAC-SNCA) mice display sex- and gene-dose-dependent phenotypic disturbances.

Journal: Behavioural brain research

Published: May 31, 2023

Immunohistochemical Demonstration of the pGlu79 α-Synuclein Fragment in Alzheimer's Disease and Its Tg2576 Mouse Model.

Immunohistochemical Demonstration of the pGlu79 α-Synuclein Fragment in Alzheimer's Disease and Its Tg2576 Mouse Model.

Journal: Biomolecules

Published: May 27, 2022

Alpha synuclein processing by MMP-3 - implications for synucleinopathies.

Alpha synuclein processing by MMP-3 - implications for synucleinopathies.

Journal: Behavioural brain research

Published: February 23, 2022

A glutaminyl cyclase-catalyzed α-synuclein modification identified in human synucleinopathies.

A glutaminyl cyclase-catalyzed α-synuclein modification identified in human synucleinopathies.

Journal: Acta neuropathologica

Published: June 11, 2021

Targeting isoaspartate-modified Aβ rescues behavioral deficits in transgenic mice with Alzheimer's disease-like pathology.

Targeting isoaspartate-modified Aβ rescues behavioral deficits in transgenic mice with Alzheimer's disease-like pathology.

Journal: Alzheimer's research & therapy

Published: May 07, 2020

Amyloid-Beta Peptides Trigger Aggregation of Alpha-Synuclein In Vitro.

Amyloid-Beta Peptides Trigger Aggregation of Alpha-Synuclein In Vitro.

Journal: Molecules (Basel, Switzerland)

Published: December 23, 2019

Endogenous mouse huntingtin is highly abundant in cranial nerve nuclei, co-aggregates to Abeta plaques and is induced in reactive astrocytes in a transgenic mouse model of Alzheimer's disease.

Endogenous mouse huntingtin is highly abundant in cranial nerve nuclei, co-aggregates to Abeta plaques and is induced in reactive astrocytes in a transgenic mouse model of Alzheimer's disease.

Journal: Acta neuropathologica communications

Published: April 16, 2019

QUINT: Workflow for Quantification and Spatial Analysis of Features in Histological Images From Rodent Brain.

QUINT: Workflow for Quantification and Spatial Analysis of Features in Histological Images From Rodent Brain.

Journal: Frontiers in neuroinformatics

Published: February 11, 2019

Cell Type-Specific Human APP Transgene Expression by Hippocampal Interneurons in the Tg2576 Mouse Model of Alzheimer's Disease.

Cell Type-Specific Human APP Transgene Expression by Hippocampal Interneurons in the Tg2576 Mouse Model of Alzheimer's Disease.

Journal: Frontiers in neuroscience

Published: December 11, 2018

Dipeptidyl-Peptidase Activity of Meprin β Links N-truncation of Aβ with Glutaminyl Cyclase-Catalyzed pGlu-Aβ Formation.

Dipeptidyl-Peptidase Activity of Meprin β Links N-truncation of Aβ with Glutaminyl Cyclase-Catalyzed pGlu-Aβ Formation.

Journal: Journal of Alzheimer's disease : JAD

Published: October 16, 2018

Showing 1-12 of 21

Last Updated: 09/26/2026