Maja T. Tomicic

Obere Zahlbacher Str. 67
Mainz, RP 55131, Germany

Last Updated: 09/26/2026

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Found 30 publications

Cellular Senescence Triggered by Food and Environmental Genotoxins.

Cellular Senescence Triggered by Food and Environmental Genotoxins.

Journal: International journal of molecular sciences

Published: January 30, 2026

Corrigendum to: "The cell cycle inhibitor p21CIP1 is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecan" [Biomed. Pharmacother. 181 (2024) 117634].

Corrigendum to: "The cell cycle inhibitor p21CIP1 is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecan" [Biomed. Pharmacother. 181 (2024) 117634].

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: January 28, 2026

Points of Departure for BPDE-induced cellular senescence and cell death are characterized by altered DNA damage signaling and regulated by unrepaired DNA double-strand breaks.

Points of Departure for BPDE-induced cellular senescence and cell death are characterized by altered DNA damage signaling and regulated by unrepaired DNA double-strand breaks.

Journal: Archives of toxicology

Published: November 27, 2025

Therapy-induced senescence of glioblastoma cells is determined by the p21CIP1-CDK1/2 axis and does not require activation of DREAM.

Therapy-induced senescence of glioblastoma cells is determined by the p21CIP1-CDK1/2 axis and does not require activation of DREAM.

Journal: Cell death & disease

Published: November 04, 2024

The cell cycle inhibitor p21CIP1 is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecan.

The cell cycle inhibitor p21CIP1 is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecan.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: August 14, 2024

The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence.

The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence.

Journal: Nucleic acids research

Published: November 09, 2023

Carboplatin-induced upregulation of pan β-tubulin and class III β-tubulin is implicated in acquired resistance and cross-resistance of ovarian cancer.

Carboplatin-induced upregulation of pan β-tubulin and class III β-tubulin is implicated in acquired resistance and cross-resistance of ovarian cancer.

Journal: Cellular and molecular life sciences : CMLS

Published: April 24, 2023

Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go?

Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go?

Journal: Cancers

Published: April 12, 2022

Epigenetic Alterations Upstream and Downstream of p53 Signaling in Colorectal Carcinoma.

Epigenetic Alterations Upstream and Downstream of p53 Signaling in Colorectal Carcinoma.

Journal: Cancers

Published: June 24, 2021

Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment.

Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment.

Journal: Cancers

Published: June 15, 2021

Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation.

Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation.

Journal: Cancers

Published: March 24, 2021

Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination.

Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination.

Journal: Cellular and molecular life sciences : CMLS

Published: January 25, 2021

Showing 1-12 of 30

Last Updated: 09/26/2026