Makoto Shimozawa

Solna, AB, Sweden

Last Updated: 09/26/2026

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Found 14 publications

Somatostatin receptor subtypes 1 and 4 regulate neprilysin, the major amyloid-β degrading enzyme in brain.

Somatostatin receptor subtypes 1 and 4 regulate neprilysin, the major amyloid-β degrading enzyme in brain.

Journal: Journal of Alzheimer's disease : JAD

Published: November 19, 2025

Effects of gonadectomy on brain sex hormone levels and amyloid pathology in male and female AppNL-G-F and AppNL-F mice.

Effects of gonadectomy on brain sex hormone levels and amyloid pathology in male and female AppNL-G-F and AppNL-F mice.

Journal: Journal of neuroendocrinology

Published: October 06, 2025

Reactive Astrocytes with Reduced Function of Glutamate Transporters in the AppNL-G-F Knock-in Mice.

Reactive Astrocytes with Reduced Function of Glutamate Transporters in the AppNL-G-F Knock-in Mice.

Journal: ACS chemical neuroscience

Published: May 27, 2025

How close is autophagy-targeting therapy for Alzheimer's disease to clinical use? A summary of autophagy modulators in clinical studies.

How close is autophagy-targeting therapy for Alzheimer's disease to clinical use? A summary of autophagy modulators in clinical studies.

Journal: Frontiers in cell and developmental biology

Published: October 31, 2024

Inhibition of Autophagy Alters Intracellular Transport of APP Resulting in Increased APP Processing.

Inhibition of Autophagy Alters Intracellular Transport of APP Resulting in Increased APP Processing.

Journal: Traffic (Copenhagen, Denmark)

Published: September 24, 2023

Identification of glycolytic proteins as binding partners of Bri2 BRICHOS domain.

Identification of glycolytic proteins as binding partners of Bri2 BRICHOS domain.

Journal: Journal of pharmaceutical and biomedical analysis

Published: April 05, 2023

The Expression of the Endocannabinoid Receptors CB2 and GPR55 Is Highly Increased during the Progression of Alzheimer's Disease in AppNL-G-F Knock-In Mice.

The Expression of the Endocannabinoid Receptors CB2 and GPR55 Is Highly Increased during the Progression of Alzheimer's Disease in AppNL-G-F Knock-In Mice.

Journal: Biology

Published: March 15, 2023

Identification of cytoskeletal proteins as binding partners of Bri2 BRICHOS domain.

Identification of cytoskeletal proteins as binding partners of Bri2 BRICHOS domain.

Journal: Molecular and cellular neurosciences

Published: January 11, 2023

Autophagy Impairment in App Knock-in Alzheimer's Model Mice.

Autophagy Impairment in App Knock-in Alzheimer's Model Mice.

Journal: Frontiers in aging neuroscience

Published: February 17, 2022

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models.

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models.

Journal: Molecular psychiatry

Published: January 17, 2022

An App knock-in rat model for Alzheimer's disease exhibiting Aβ and tau pathologies, neuronal death and cognitive impairments.

An App knock-in rat model for Alzheimer's disease exhibiting Aβ and tau pathologies, neuronal death and cognitive impairments.

Journal: Cell research

Published: April 03, 2021

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.

Journal: Autophagy

Published: February 26, 2021

Showing 1-12 of 14

Last Updated: 09/26/2026