Mara Caputo
Goeteborg, O, Sweden
Last Updated: 09/26/2026
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Found 14 publications
Inhibition of GCKIII kinases STK25 and MST3 mitigates organ lipotoxicity and enhances metabolic resilience under nutritional stress.
Journal: BMC medicine
Published: March 12, 2025
Therapeutic Potential of STE20-Type Kinase STK25 Inhibition for the Prevention and Treatment of Metabolically Induced Hepatocellular Carcinoma.
Journal: Cellular and molecular gastroenterology and hepatology
Published: November 12, 2024
GCKIII kinases control hepatocellular lipid homeostasis via shared mode of action.
Journal: Journal of lipid research
Published: June 27, 2024
Genetic Ablation of STE20-Type Kinase MST4 Does Not Alleviate Diet-Induced MASLD Susceptibility in Mice.
Journal: International journal of molecular sciences
Published: January 23, 2024
Knockout of STE20-type kinase TAOK3 does not attenuate diet-induced NAFLD development in mice.
Journal: Molecular medicine (Cambridge, Mass.)
Published: August 02, 2023
GCKIII kinases in lipotoxicity: Roles in NAFLD and beyond.
Journal: Hepatology communications
Published: February 24, 2022
Inhibition of MAP4K4 signaling initiates metabolic reprogramming to protect hepatocytes from lipotoxic damage.
Journal: Journal of lipid research
Published: February 14, 2022
STE20-type kinase TAOK3 regulates hepatic lipid partitioning.
Journal: Molecular metabolism
Published: August 18, 2021
Silencing of STE20-type kinase STK25 in human aortic endothelial and smooth muscle cells is atheroprotective.
Journal: Communications biology
Published: August 16, 2021
Antagonizing STK25 Signaling Suppresses the Development of Hepatocellular Carcinoma Through Targeting Metabolic, Inflammatory, and Pro-Oncogenic Pathways.
Journal: Cellular and molecular gastroenterology and hepatology
Published: April 22, 2021
STE20-Type Protein Kinase MST4 Controls NAFLD Progression by Regulating Lipid Droplet Dynamics and Metabolic Stress in Hepatocytes.
Journal: Hepatology communications
Published: November 11, 2020
Last Updated: 09/26/2026