Marcia Faria

Lisbon, 1649016, Portugal

Last Updated: 09/26/2026

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Found 9 publications

Adherens Junction Integrity Is a Critical Determinant of Sodium Iodide Symporter Residency at the Plasma Membrane of Thyroid Cells.

Adherens Junction Integrity Is a Critical Determinant of Sodium Iodide Symporter Residency at the Plasma Membrane of Thyroid Cells.

Journal: Cancers
Published: August 16, 2022

MAPK Inhibition Requires Active RAC1 Signaling to Effectively Improve Iodide Uptake by Thyroid Follicular Cells.

MAPK Inhibition Requires Active RAC1 Signaling to Effectively Improve Iodide Uptake by Thyroid Follicular Cells.

Journal: Cancers
Published: November 08, 2021

Analysis of NIS Plasma Membrane Interactors Discloses Key Regulation by a SRC/RAC1/PAK1/PIP5K/EZRIN Pathway with Potential Implications for Radioiodine Re-Sensitization Therapy in Thyroid Cancer.

Analysis of NIS Plasma Membrane Interactors Discloses Key Regulation by a SRC/RAC1/PAK1/PIP5K/EZRIN Pathway with Potential Implications for Radioiodine Re-Sensitization Therapy in Thyroid Cancer.

Journal: Cancers
Published: September 28, 2021

Inflammatory Microenvironment Modulation of Alternative Splicing in Cancer: A Way to Adapt.

Inflammatory Microenvironment Modulation of Alternative Splicing in Cancer: A Way to Adapt.

Journal: Advances in experimental medicine and biology
Published: March 05, 2020

TNFα-mediated activation of NF-κB downregulates sodium-iodide symporter expression in thyroid cells.

TNFα-mediated activation of NF-κB downregulates sodium-iodide symporter expression in thyroid cells.

Journal: PloS one
Published: November 06, 2019

Antagonistic effects of RAC1 and tumor-related RAC1b on NIS expression in thyroid.

Antagonistic effects of RAC1 and tumor-related RAC1b on NIS expression in thyroid.

Journal: Journal of molecular endocrinology
Published: September 08, 2019

Prolonged co-treatment with HGF sustains epithelial integrity and improves pharmacological rescue of Phe508del-CFTR.

Prolonged co-treatment with HGF sustains epithelial integrity and improves pharmacological rescue of Phe508del-CFTR.

Journal: Scientific reports
Published: May 14, 2018

RAC1b overexpression stimulates proliferation and NF-kB-mediated anti-apoptotic signaling in thyroid cancer cells.

RAC1b overexpression stimulates proliferation and NF-kB-mediated anti-apoptotic signaling in thyroid cancer cells.

Journal: PloS one
Published: August 22, 2016

Extending the Impact of RAC1b Overexpression to Follicular Thyroid Carcinomas.

Extending the Impact of RAC1b Overexpression to Follicular Thyroid Carcinomas.

Journal: International journal of endocrinology
Published: January 14, 2016
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Last Updated: 09/26/2026