Last Updated: 09/26/2026
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Found 9 publications
Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling.
Journal: Cardiovascular research
Published: August 02, 2016
Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice.
Journal: Circulation. Heart failure
Published: November 23, 2014
Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.
Journal: American journal of physiology. Heart and circulatory physiology
Published: August 17, 2014
CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans.
Journal: Cardiovascular research
Published: June 04, 2014
Thrombospondin-1 induction in the diabetic myocardium stabilizes the cardiac matrix in addition to promoting vascular rarefaction through angiopoietin-2 upregulation.
Journal: Circulation research
Published: October 02, 2013
Systematic characterization of myocardial inflammation, repair, and remodeling in a mouse model of reperfused myocardial infarction.
Journal: The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society
Published: May 30, 2013
Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts.
Journal: Arteriosclerosis, thrombosis, and vascular biology
Published: September 22, 2012
The extracellular matrix modulates fibroblast phenotype and function in the infarcted myocardium.
Journal: Journal of cardiovascular translational research
Published: July 02, 2012
Endogenous thrombospondin 1 protects the pressure-overloaded myocardium by modulating fibroblast phenotype and matrix metabolism.
Journal: Hypertension (Dallas, Tex. : 1979)
Published: September 28, 2011
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Last Updated: 09/26/2026